F
Fergus Shanahan
Researcher at National University of Ireland
Publications - 727
Citations - 59181
Fergus Shanahan is an academic researcher from National University of Ireland. The author has contributed to research in topics: Inflammatory bowel disease & Gut flora. The author has an hindex of 117, co-authored 705 publications receiving 51963 citations. Previous affiliations of Fergus Shanahan include Imperial College London & Mater Misericordiae Hospital.
Papers
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Journal ArticleDOI
Effect of probiotic and vitamin D supplementation on markers of vitamin D status and bone turnover in healthy adults
Tom R. Hill,Lorraine Brennan,A. O'Connor,L. Scully,S. Healy,Aifric O'Sullivan,B. Mion,G. Dawson,S. Kaluskar,Michael J. Gibney,Fergus Shanahan,Kevin D. Cashman +11 more
TL;DR: The objective of the present study was to examine the effect of 4-week supplementation with probiotic bacteria and vitamin D on serum 25-hydroxyvitamin D (S-25(OH)D), parathyroid hormone (PTH) and biochemical markers of bone turnover in healthy adults.
Journal ArticleDOI
Appropriateness of laboratory tests: requests for atypical pneumonia serology in a teaching hospital.
TL;DR: Most requests for serology for organisms causing atypical pneumonia were inappropriate and in the majority of cases the test was incorrectly used.
Journal ArticleDOI
Regulation of Myelination in the Prefrontal Cortex by the Gut Microbiota: Implications for Health and Disease
Alan E. Hoban,Roman M. Stilling,Lieve Desbonnet,Fergus Shanahan,Timothy G. Dinan,John F. Cryan,Gerard Clarke +6 more
TL;DR: A large number of studies have suggested that the gut microbiome plays a role in multiple sclerosis, but the role of these microbes and their role in disease is still poorly understood.
Journal ArticleDOI
Fecal microbiota-based treatment for recurrent Clostridioides difficile infection
Jens Walter,Fergus Shanahan +1 more
TL;DR: Rebyota as mentioned in this paper is a rectally administered fecal microbiota suspension for prevention of recurrence of Clostridioides difficile infection, which probably involves competitive exclusion of C. diffice by donor microbes with reduced toxin production; other factors may include restoration of protective taxa and modulation of the recipient's microbiome by phage, donor microbes, or metabolites.