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Garry P. Nolan

Researcher at Stanford University

Publications -  519
Citations -  54521

Garry P. Nolan is an academic researcher from Stanford University. The author has contributed to research in topics: Immune system & T cell. The author has an hindex of 104, co-authored 474 publications receiving 46025 citations. Previous affiliations of Garry P. Nolan include Massachusetts Institute of Technology & New York University.

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Journal ArticleDOI

Mass cytometry: The time to settle down.

TL;DR: This special Cytometry Part A issue includes the first OMIP describing a mass cytometry panel for the immune phenotype of human peripheral leukocytes together with a series of manuscripts introducing new reagents, protocols, quality controls and, also, a nice example of how the multidimensional nature ofmass cytometry can address important biological question such as the status of a “challenged” immune system.
Journal ArticleDOI

Spatial Epitope Barcoding Reveals Subclonal Tumor Patch Behaviors

TL;DR: EpicTags was developed, which was coupled to multiplex ion beam imaging (EpicMIBI) for in situ tracking of barcodes within tissue microenvironments, and can facilitate in situ interrogation of cell-intrinsic and cell-extrinsics processes involved in intratumoral heterogeneity.
Peer ReviewDOI

Author response: The Human Cell Atlas

Aviv Regev, +81 more
- 20 Nov 2017 - 
Journal ArticleDOI

WebFlow: a software package for high-throughput analysis of flow cytometry data.

TL;DR: WebFlow provides a novel platform for quantitative analysis of flow cytometric data from high-throughput drug screening or disease profiling experiments, and enables plate-based annotation of large data sets, which provides the basis for exploratory data analysis tools and rapid visualization of multiple different parameters.
Journal ArticleDOI

EBI3 regulates the NK cell response to mouse cytomegalovirus infection

TL;DR: EBI3 dampens the immune response against MCMV infection, resulting in prolonged viral persistence, and provides insight into how CMV establishes latent infection.