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Khalid Abdul Kadir

Researcher at Monash University Malaysia Campus

Publications -  64
Citations -  4337

Khalid Abdul Kadir is an academic researcher from Monash University Malaysia Campus. The author has contributed to research in topics: Diabetes mellitus & Metabolic syndrome. The author has an hindex of 20, co-authored 62 publications receiving 3523 citations. Previous affiliations of Khalid Abdul Kadir include Monash University & National University of Malaysia.

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Journal ArticleDOI

Worldwide trends in diabetes since 1980: a pooled analysis of 751 population-based studies with 4.4 million participants

Bin Zhou, +497 more
- 09 Apr 2016 - 
TL;DR: In this article, the authors used a Bayesian hierarchical model to estimate trends in diabetes prevalence, defined as fasting plasma glucose of 7.0 mmol/L or higher, or history of diagnosis with diabetes, or use of insulin or oral hypoglycaemic drugs in 200 countries and territories in 21 regions, by sex and from 1980 to 2014.
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Effects of diabetes definition on global surveillance of diabetes prevalence and diagnosis: A pooled analysis of 96 population-based studies with 331 288 participants

Goodarz Danaei, +430 more
TL;DR: The effect of different diagnostic definitions on both the population prevalence of diabetes and the classification of previously undiagnosed individuals as having diabetes versus not having diabetes in a pooled analysis of data from population-based health examination surveys in different regions is assessed.
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Stress and Its Effects on Glucose Metabolism and 11β-HSD Activities in Rats Fed on a Combination of High-Fat and High-Sucrose Diet with Glycyrrhizic Acid.

TL;DR: GA-induced improvements in 11β-HSD activities in certain tissues were observed, although it is uncertain if these effects are manifested after adaptation due to the withdrawal of the stress response, and the ability of GA to improve stress-induced disturbances in the absence of adaptation needs to be investigated further.
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Glycyrrhizic acid improved lipoprotein lipase expression, insulin sensitivity, serum lipid and lipid deposition in high-fat diet-induced obese rats.

TL;DR: GA may be a potential compound in improving dyslipidaemia by selectively inducing LPL expression in non-hepatic tissues and was accompanied by a GA-mediated improvement in insulin sensitivity, which may be associated with a decrease in tissue lipid deposition.