L
Lewis L. Lanier
Researcher at University of California, San Francisco
Publications - 576
Citations - 93495
Lewis L. Lanier is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: Interleukin 21 & Natural killer cell. The author has an hindex of 159, co-authored 554 publications receiving 86677 citations. Previous affiliations of Lewis L. Lanier include University of Rome Tor Vergata & Cancer Research Institute.
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Journal ArticleDOI
Transplantable b-cell lymphomas in b10.h-2ah-4bp/wts Mice.
Lewis L. Lanier,Lewis L. Lanier,Larry W. Arnold,Larry W. Arnold,Richard B. Raybourne,Richard B. Raybourne,Stephen T. Russell,Stephen T. Russell,Michael A. Lynes,Michael A. Lynes,Noel L. Warnert,Noel L. Warnert,Geoffrey Haughton,Geoffrey Haughton +13 more
TL;DR: The characterization of a series of tumors induced and established in this "double congenic" strain of mice, including a T-lymphoma and a macrophage tumor, are shown.
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NKG2C Natural Killer Cells in Bronchoalveolar Lavage Are Associated With Cytomegalovirus Viremia and Poor Outcomes in Lung Allograft Recipients.
Daniel R Calabrese,Tiffany Chong,Angelia Wang,Jonathan P. Singer,M. Gottschall,Steven R. Hays,Jeffrey A. Golden,Jasleen Kukreja,Lewis L. Lanier,Qizhi Tang,John R. Greenland +10 more
TL;DR: The BAL NKG2C+ NK cell proportion may be a relevant biomarker for assessing risk of CMV viremia and quantifying potential CMV-related graft injury that can lead to CLAD or death.
Journal ArticleDOI
The gamma T-cell antigen receptor.
Lewis L. Lanier,Andrew T. Serafini,Joyce J. Ruitenberg,Steve Cwirla,Nancy A. Federspiel,Joseph H. Phillips,James P. Allison,Arthur Weiss +7 more
TL;DR: Antibodies against CD3 or gamma-TCR can induce proliferation and IL-2 secretion and can either augment or inhibit cytotoxicity, demonstrating that the gamma/delta- TCR is a functional receptor.
Journal ArticleDOI
T-cell costimulation via CD28-CD80/CD86 and CD40-CD40 ligand interactions
Chamorro Somoza,Lewis L. Lanier +1 more
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NK Cells Are Not Required for Spontaneous Autoimmune Diabetes in NOD Mice
Joshua N. Beilke,Craig T. Meagher,Karoline A. Hosiawa,Marine Champsaur,Jeffrey A. Bluestone,Lewis L. Lanier +5 more
TL;DR: In spontaneous disease, the deletion of NK cells had no significant impact on disease onset and NK cells were also not required to promote disease induced by adoptively transferred pathogenic CD4+ T cells, thus, NK cells are not required for spontaneous autoimmune diabetes in NOD mice.