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Patrick L. McGeer
Researcher at University of British Columbia
Publications - 569
Citations - 61292
Patrick L. McGeer is an academic researcher from University of British Columbia. The author has contributed to research in topics: Microglia & Alzheimer's disease. The author has an hindex of 122, co-authored 569 publications receiving 58584 citations. Previous affiliations of Patrick L. McGeer include Laval University & Kyoto University.
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Journal ArticleDOI
On the survival time of a tangled neuron in the hippocampal CA4 region in parkinsonism dementia complex of Guam.
TL;DR: It is concluded that tangle development in CA4 commenced close to the onset of the disease, and the average lifetime of tangled neurons does not alter as the disease progresses.
Book ChapterDOI
Signaling in the Nervous System
TL;DR: Chapter 1 was devoted to giving an account of what a neuron is in itself, and now this chapter comes to consider how neurons are concerned in receiving and in giving signals.
Journal ArticleDOI
Expression of complement messenger RNAs by human endothelial cells.
TL;DR: The results indicate that endothelial cells may be a source of complement proteins in brain and other organs of the body.
Journal ArticleDOI
Transplantation of cultured human spinal cord cells into the rat motor cortex: use of Phaseolus vulgaris leucoagglutinin as a cell marker.
TL;DR: These findings extend the previous studies in which it was shown that cultured human fetal adrenal medulla and sympathetic ganglia cells could be successfully transplanted to non-immunosuppressed rat brain and suggest that PHA may be a valuable marker for transplanted cells at least for 1.5 months post-transplantation.
Journal ArticleDOI
Two types of spheroid bodies in the nigral neurons in Parkinson's disease.
TL;DR: Dendritic spheroid bodies and Lewy bodies identified in comparable numbers in the substantia nigra pars compacta of nine parkinsonian cases and one case of striatonigral degeneration but were not found irt cases of Huntington's disease or neurologically normal controls indicate that dendritic SBs and extracellular LBs may have a common molecular pathogenetic origin in Parkinson's disease.