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Journal ArticleDOI

The role of iodine in human growth and development.

Michael B. Zimmermann
- 01 Aug 2011 - 
- Vol. 22, Iss: 6, pp 645-652
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TLDR
Iodine prophylaxis of deficient populations with periodic monitoring is an extremely cost effective approach to reduce the substantial adverse effects of iodine deficiency throughout the life cycle.
About
This article is published in Seminars in Cell & Developmental Biology.The article was published on 2011-08-01. It has received 295 citations till now. The article focuses on the topics: Iodine deficiency & Cretinism.

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Citations
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A Scoping Review of Iodine and Fluoride in Pregnancy in Relation to Maternal Thyroid Function and Offspring Neurodevelopment

TL;DR: In this article , the authors provide a scoping review of the literature on I and F exposure during pregnancy and their individual effects on thyroid function and offspring neurodevelopment and conclude more studies are needed.
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Urinary iodine concentration and its associations with thyroid function in pregnant women of Shanghai

TL;DR: In this article , a cross-sectional study was conducted to assess the iodine status and its associations with thyroid function in pregnant women of Shanghai, where participants were divided into four groups based on their UIC values and a correlation analysis was used to investigate the association between UIC and thyroid functionassociated parameters.
Journal Article

Iodine status and thyroid volume among school children: approach in methodology of sarawak iodine deficiency disorder survey 2018

TL;DR: The approach in methodology of Sarawak IDD Survey 2018 which aimed to determine the current status of IDD among school children in SarawK after 10 years of USI implementation will highlight the impact of mandatory USI towards the iodine level among schoolChildren in Sarawi.
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The correlation between iodine nutrition and autoimmunity among hyperthyroid goiter patients in tertiary care hospital of Tamil Nadu

TL;DR: There were excess UIEs among patients; hence, these associated complications, namely, benign goiter (14%), thyroiditis (43%), cancer of thyroid (4%), and thyrotoxicosis (39%) were observed.
References
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Dietary reference intakes: vitamin A, vitamin K, arsenic, boron, chromium, copper, iodine, iron, manganese, molybdenum, nickel, silicon, vanadium, and zinc.

TL;DR: The DRIs represent the new approach adopted by the Food and Nutrition Board to providing quantitative estimates of nutrient intakes for use in a variety of settings, replacing and expanding on the past 50 years of periodic updates and revisions of the Recommended Dietary Allowances.
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Insulin-Like Growth Factors and Their Binding Proteins: Biological Actions*

TL;DR: In recognition of its generalized pleiotypic actions, sulfation factor was renamed somatomedin (mediator of the effects of somatotropin) and was included in the emerging classification of broad spectrum growth factors along with platelet derived growth factor, fibroblast growth factors, and epidermal growth factor.
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Maternal thyroid deficiency during pregnancy and subsequent neuropsychological development of the child.

TL;DR: Undiagnosed hypothyroidism in pregnant women may adversely affect their fetuses; therefore, screening for thyroid deficiency during pregnancy may be warranted.
Book

Dietary reference intakes for vitamin A, vitamin K, arsenic, boron, chromium, copper, iodine, iron, manganese, molybdenum, nickel, silicon, vanadium, and zinc : a report of the Panel on Micronutrients, Subcommittees on Upper Reference Levels of Nutrients and of Interpretation and Use of Dietary Reference Intakes, and the Standing Committee on the Scientific Evaluation of Dietary Reference Intakes, Food and Nutrition Board, Institute of Medicine

TL;DR: In this article, a review of the scientific literature regarding dietary micronutrients, recommendations have been formulated regarding vitamins A and K, iron, iodine, chromium, copper, manganese, molybdenum, zinc, and other potentially beneficial trace elements such as boron to determine the roles, if any, they play in health.
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Impaired spatial learning in alpha-calcium-calmodulin kinase II mutant mice

TL;DR: The data considerably strengthen the contention that the synaptic changes exhibited in LTP are the basis for spatial memory.
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