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Journal ArticleDOI

The role of iodine in human growth and development.

Michael B. Zimmermann
- 01 Aug 2011 - 
- Vol. 22, Iss: 6, pp 645-652
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TLDR
Iodine prophylaxis of deficient populations with periodic monitoring is an extremely cost effective approach to reduce the substantial adverse effects of iodine deficiency throughout the life cycle.
About
This article is published in Seminars in Cell & Developmental Biology.The article was published on 2011-08-01. It has received 295 citations till now. The article focuses on the topics: Iodine deficiency & Cretinism.

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Citations
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Journal ArticleDOI

Effects of Iodized Salt and Iodine Supplements on Prenatal and Postnatal Growth: A Systematic Review.

TL;DR: A systematic review of iodine fortification or supplementation on prenatal and postnatal growth outcomes in noncretinous children identified few well-designed trials examining the effects of iodine repletion on growth, and it is uncertain whether prenatal iodineRepletion increases infant growth.
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Consequences of Severe Iodine Deficiency in Pregnancy: Evidence in Humans.

TL;DR: The impact of severe iodine deficiency on maternofetal, neonatal, and offspring outcomes is reviewed and its epidemiology, classification of iodine deficiency severity, and current recommendations to prevent iodine deficiency in childbearing age and pregnant women are discussed.
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Impact of Endocrine Disruptors on the Thyroid Hormone System.

TL;DR: The thyroid hormone (TH) system plays a central role in central physiological processes of many species, including mammals and humans, ranging from growth and cell differentiation, energy metabolism, thermoregulation and phasing of hibernation or annual movements of migratory species, metamorphosis from larvae to adult forms, brain development, reproduction, or the cardiovascular system.
Journal ArticleDOI

Fluoride Sources, Toxicity and Its Amelioration: A Review

TL;DR: Fl uoride (F) presents in the halogenated group of the periodic table and has the characteristics of electronegativity and exerts adverse effects mainly through the attenuation of antioxidant defense mechanism and chelation of enzymatic cofactors.
References
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Journal ArticleDOI

Dietary reference intakes: vitamin A, vitamin K, arsenic, boron, chromium, copper, iodine, iron, manganese, molybdenum, nickel, silicon, vanadium, and zinc.

TL;DR: The DRIs represent the new approach adopted by the Food and Nutrition Board to providing quantitative estimates of nutrient intakes for use in a variety of settings, replacing and expanding on the past 50 years of periodic updates and revisions of the Recommended Dietary Allowances.
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Insulin-Like Growth Factors and Their Binding Proteins: Biological Actions*

TL;DR: In recognition of its generalized pleiotypic actions, sulfation factor was renamed somatomedin (mediator of the effects of somatotropin) and was included in the emerging classification of broad spectrum growth factors along with platelet derived growth factor, fibroblast growth factors, and epidermal growth factor.
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Maternal thyroid deficiency during pregnancy and subsequent neuropsychological development of the child.

TL;DR: Undiagnosed hypothyroidism in pregnant women may adversely affect their fetuses; therefore, screening for thyroid deficiency during pregnancy may be warranted.
Book

Dietary reference intakes for vitamin A, vitamin K, arsenic, boron, chromium, copper, iodine, iron, manganese, molybdenum, nickel, silicon, vanadium, and zinc : a report of the Panel on Micronutrients, Subcommittees on Upper Reference Levels of Nutrients and of Interpretation and Use of Dietary Reference Intakes, and the Standing Committee on the Scientific Evaluation of Dietary Reference Intakes, Food and Nutrition Board, Institute of Medicine

TL;DR: In this article, a review of the scientific literature regarding dietary micronutrients, recommendations have been formulated regarding vitamins A and K, iron, iodine, chromium, copper, manganese, molybdenum, zinc, and other potentially beneficial trace elements such as boron to determine the roles, if any, they play in health.
Journal ArticleDOI

Impaired spatial learning in alpha-calcium-calmodulin kinase II mutant mice

TL;DR: The data considerably strengthen the contention that the synaptic changes exhibited in LTP are the basis for spatial memory.
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