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Adeline Vanderver

Researcher at Children's Hospital of Philadelphia

Publications -  207
Citations -  9841

Adeline Vanderver is an academic researcher from Children's Hospital of Philadelphia. The author has contributed to research in topics: Leukodystrophy & Leukoencephalopathy. The author has an hindex of 42, co-authored 172 publications receiving 7751 citations. Previous affiliations of Adeline Vanderver include Children's National Medical Center & Columbia University.

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Mutations in ADAR1 cause Aicardi-Goutières syndrome associated with a type I interferon signature

Gillian I. Rice, +53 more
- 01 Nov 2012 - 
TL;DR: It is shown that mutations in ADAR1 cause the autoimmune disorder Aicardi-Goutières syndrome (AGS), and it is speculated that ADar1 may limit the cytoplasmic accumulation of the dsRNA generated from genomic repetitive elements.
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Mutations involved in Aicardi-Goutières syndrome implicate SAMHD1 as regulator of the innate immune response.

Gillian I. Rice, +50 more
- 14 Jun 2009 - 
TL;DR: Mutations in SAMHD1 are described as the cause of Aicardi-Goutières syndrome at the AGS5 locus and data is presented to show that SAM HD1 may act as a negative regulator of the cell-intrinsic antiviral response.
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Gain-of-function mutations in IFIH1 cause a spectrum of human disease phenotypes associated with upregulated type I interferon signaling

Gillian I. Rice, +56 more
- 01 May 2014 - 
TL;DR: It is demonstrated that aberrant sensing of nucleic acids can cause immune upregulation and heterozygous mutations in the cytosolic double-stranded RNA receptor gene IFIH1 (also called MDA5) cause a spectrum of neuroimmunological features consistently associated with an enhanced interferon state.
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Characterization of human disease phenotypes associated with mutations in TREX1, RNASEH2A, RNASEH2B, RNASEH2C, SAMHD1, ADAR, and IFIH1.

Yanick J. Crow, +135 more
TL;DR: A robust relationship between mutations in all seven genes with increased type I interferon activity in cerebrospinal fluid and serum, and the increased expression of interferOn‐stimulated gene transcripts in peripheral blood is observed.
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Clinical and Molecular Phenotype of Aicardi-Goutières Syndrome

Gillian I. Rice, +117 more
TL;DR: The analysis defines the phenotypic spectrum of AGS and suggests a coherent mutation-screening strategy in this heterogeneous disorder, and indicates that at least one further AGS-causing gene remains to be identified.