E
Erwin W. Gelfand
Researcher at University of Colorado Denver
Publications - 679
Citations - 37565
Erwin W. Gelfand is an academic researcher from University of Colorado Denver. The author has contributed to research in topics: Immunoglobulin E & T cell. The author has an hindex of 99, co-authored 675 publications receiving 36059 citations. Previous affiliations of Erwin W. Gelfand include University of Colorado Hospital & University of Virginia.
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Hepatotoxicity with encephalopathy associated with aspirin therapy in rheumatoid arthritis
TL;DR: Acute hepatic decompensation and encephalopathy may occur as a consequence of aspirin hepatotoxicity in JRA and justify sequential observations of liver function tests and salicylate levels in such patients.
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Rearrangement of the T cell receptor gamma-chain gene in childhood acute lymphoblastic leukemia
TL;DR: The combined analysis of rearrangement patterns of IgH, T beta, and T gamma genes provides new criteria for defining the cellular origin of leukemic cells and for further delineation of leukemia cell heterogeneity.
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Requirement for the p75 TNF-α Receptor 2 in the Regulation of Airway Hyperresponsiveness by γδ T Cells
Arihiko Kanehiro,Michael Lahn,Mika J. Mäkelä,Azzeddine Dakhama,Anthony Joetham,Yeong Ho Rha,Willi K. Born,Erwin W. Gelfand +7 more
TL;DR: Data indicate that, in the absence of TNFR1 (p55), where TNF-α uses the p75 pathway exclusively, the development of AHR is regulated by γδ T cells.
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IL-2 and IL-18 Attenuation of Airway Hyperresponsiveness Requires STAT4, IFN-γ, and Natural Killer Cells
Shigeki Matsubara,Katsuyuki Takeda,Taku Kodama,Anthony Joetham,Nobuaki Miyahara,Toshiyuki Koya,Christina H. Swasey,Masakazu Okamoto,Azzeddine Dakhama,Erwin W. Gelfand +9 more
TL;DR: It is demonstrated that the combination of IL-2 and IL-18 prevents AHR and airway inflammation, likely through IL-12-mediated induction of IFN-gamma production in NK cells.
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Plasticity of Regulatory T Cells: Subversion of Suppressive Function and Conversion to Enhancement of Lung Allergic Responses
Anthony Joetham,Shigeki Matsubara,Masakazu Okamoto,Katsuyuki Takeda,Nobuaki Miyahara,Azzeddine Dakhama,Erwin W. Gelfand +6 more
TL;DR: The regulatory function of nTregs can be subverted by reducing the expression of Foxp3 and following signaling through glucocorticoid-induced TNFR-related protein are converted nT Regs into IL-13-producing CD4+ T cells mediating lung allergic responses.