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Luisa Benussi

Researcher at University of Brescia

Publications -  551
Citations -  18162

Luisa Benussi is an academic researcher from University of Brescia. The author has contributed to research in topics: Large Hadron Collider & Frontotemporal dementia. The author has an hindex of 58, co-authored 525 publications receiving 13817 citations. Previous affiliations of Luisa Benussi include University of Verona & Sacred Heart Hospital.

Papers
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Elevated CSF and plasma complement proteins in genetic frontotemporal dementia: results from the GENFI study

Emma L. van der Ende, +145 more
- 05 Sep 2022 - 
TL;DR: In this paper , the value of complement proteins, which are key components of innate immunity, as biomarkers in cerebrospinal fluid (CSF) and plasma of presymptomatic and symptomatic genetic FTD mutation carriers was determined.
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Measurement of differential tt¯ production cross sections in the full kinematic range using lepton+jets events from proton-proton collisions at s=13 TeV

Armen Tumasyan, +2329 more
- 30 Nov 2021 - 
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Study of the A(Kstop−,π±Σ∓)A′ reaction

Michelangelo Agnello, +43 more
- 01 Apr 2010 - 
TL;DR: In this article, the authors describe a recent experimental study of the A ( K s t o p −, π ± Σ ∓ ) A reaction on light nuclei, Li 6, 7, Be 9, C 13 and O 16.

Genetics and Expression Analysis of the Specificity Protein 4 Gene (SP4) inPatients with Alzheimer's Disease and Frontotemporal Lobar Degeneration.

Chiara Villa, +20 more
TL;DR: According to these results, SP4 gene does not act as a susceptibility factor either for AD or FTLD, however, Sp4 mRNA levels are upregulated in patients, possibly resulting in an aberrant expression of downstream target genes involved in the pathogenesis of both diseases.
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Asymptomatic Carriers of Presenilin-1 E318G Variant Show no Cerebrospinal Fluid Biochemical Signs Suggestive of Alzheimer's disease in a Family with Late-onset Dementia.

Vladimiro Artuso, +8 more
- 31 Dec 2018 - 
TL;DR: A large Italian family followed with the late-onset AD and the E318G variant from January 2003 to January 2018 may help elucidate E 318G's role in AD and globally points to a null effect of this variant.