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Mireia Jordà

Researcher at Carlos III Health Institute

Publications -  32
Citations -  2751

Mireia Jordà is an academic researcher from Carlos III Health Institute. The author has contributed to research in topics: DNA methylation & Epigenetics. The author has an hindex of 13, co-authored 24 publications receiving 2459 citations.

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Insights into social insects from the genome of the honeybee Apis mellifera

George M. Weinstock, +228 more
- 26 Oct 2006 - 
TL;DR: The genome sequence of the honeybee Apis mellifera is reported, suggesting a novel African origin for the species A. melliferA and insights into whether Africanized bees spread throughout the New World via hybridization or displacement.
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Functional CpG Methylation System in a Social Insect

TL;DR: Using the recently sequenced honey bee genome, a bioinformatic, molecular, and biochemical characterization of a functional DNA methylation system in an insect is presented and catalytically active orthologs of the vertebrate DNA methyltransferases Dnmt1 and DNmt3a and b are reported.
Journal ArticleDOI

Epigenetics of host-pathogen interactions: the road ahead and the road behind.

TL;DR: The evidence available for the role epigenetics on host- Pathogen interactions, and the utility and versatility of the epigenetic technologies available that can be cross-applied to host-pathogen studies are reviewed are reviewed.
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Genome-wide tracking of unmethylated DNA Alu repeats in normal and cancer cells

TL;DR: There is an inverse relationship in Alu families with respect to their age and methylation status: the youngest elements exhibit the highest prevalence of the SmaI site but the lower rates of unmethylation, consistent with a stronger silencing pressure on the youngest repetitive elements, which are closer to genes.
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DNA methylation profiling of well‐differentiated thyroid cancer uncovers markers of recurrence free survival

TL;DR: It was found that follicular tumors had higher levels of methylation, which seemed to accumulate in a progressive manner along the tumorigenic process from adenomas to carcinomas, and etoposide‐induced 2.4 and Wilms tumor 1 as novel prognostic markers related to recurrence‐free survival.