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Tom Callens

Researcher at University of Alabama at Birmingham

Publications -  46
Citations -  2446

Tom Callens is an academic researcher from University of Alabama at Birmingham. The author has contributed to research in topics: Population & Gene. The author has an hindex of 25, co-authored 45 publications receiving 2123 citations. Previous affiliations of Tom Callens include Ghent University & Ghent University Hospital.

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Clinical spectrum of individuals with pathogenic NF1 missense variants affecting p.Met1149, p.Arg1276, and p.Lys1423: genotype–phenotype study in neurofibromatosis type 1

Magdalena Koczkowska, +107 more
- 01 Jan 2020 - 
TL;DR: Although clinically relevant genotype–phenotype correlations are rare in NF1, each affecting only a small percentage of individuals, together they impact counseling and management of a significant number of the NF1 population.
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The Development of Cutaneous Neurofibromas

TL;DR: It is speculated that the development of cutaneous neurofibromas includes the recruitment of multipotent NF1(+/-) precursor cells, which may be derived from the multipotent cells of the hair roots, which often are intimately associated with microscopic neurof fibromas.
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A stochastic movement simulator improves estimates of landscape connectivity.

TL;DR: In this article, the authors compared circuit theory and least-cost paths (LCPs) with an individual-based simulator (SMS) to predict genetic connectivity in the field of landscape connectivity.
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Expanding the clinical phenotype of individuals with a 3-bp in-frame deletion of the NF1 gene (c.2970_2972del): an update of genotype-phenotype correlation.

Magdalena Koczkowska, +69 more
- 01 Apr 2019 - 
TL;DR: It is demonstrated that individuals with the NF1 p.Met992del pathogenic variant have a mild NF1 phenotype lacking clinically suspected plexiform, cutaneous, or subcutaneous neurofibromas, however, learning difficulties are clearly part of the phenotypic presentation in these individuals and will require specialized care.
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Characterisation of two different nonsense mutations, C6792A and C6792G, causing skipping of exon 37 in the NF1 gene

TL;DR: This is the first report showing that nonsense mutations in the NF1 gene can induce the skipping of a constitutive exon, in 21 unrelated NF1 patients using reverse transcription-polymerase chain reaction and protein truncation analysis.