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Yichao Sun

Researcher at Memorial Sloan Kettering Cancer Center

Publications -  23
Citations -  34654

Yichao Sun is an academic researcher from Memorial Sloan Kettering Cancer Center. The author has contributed to research in topics: Genome & Gene. The author has an hindex of 15, co-authored 20 publications receiving 25842 citations. Previous affiliations of Yichao Sun include Baylor College of Medicine.

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Integrative analysis of complex cancer genomics and clinical profiles using the cBioPortal

TL;DR: A practical guide to the analysis and visualization features of the cBioPortal for Cancer Genomics, which makes complex cancer genomics profiles accessible to researchers and clinicians without requiring bioinformatics expertise, thus facilitating biological discoveries.
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The cancer genome atlas pan-cancer analysis project

John N. Weinstein, +379 more
- 01 Oct 2013 - 
TL;DR: The Pan-Cancer initiative compares the first 12 tumor types profiled by TCGA with a major opportunity to develop an integrated picture of commonalities, differences and emergent themes across tumor lineages.
Journal Article

The Cancer Genome Atlas Pan-Cancer analysis project

Kyle Chang, +337 more
- 01 Sep 2013 - 
TL;DR: The Cancer Genome Atlas (TCGA) Research Network has profiled and analyzed large numbers of human tumors to discover molecular aberrations at the DNA, RNA, protein and epigenetic levels as mentioned in this paper.
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Comprehensive molecular characterization of gastric adenocarcinoma

Adam J. Bass, +257 more
- 11 Sep 2014 - 
TL;DR: A comprehensive molecular evaluation of 295 primary gastric adenocarcinomas as part of The Cancer Genome Atlas (TCGA) project is described and a molecular classification dividing gastric cancer into four subtypes is proposed.
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Comprehensive genomic characterization of head and neck squamous cell carcinomas

Michael S. Lawrence, +309 more
- 29 Jan 2015 - 
TL;DR: It is shown that human-papillomavirus-associated tumours are dominated by helical domain mutations of the oncogene PIK3CA, novel alterations involving loss of TRAF3, and amplification of the cell cycle gene E2F1.