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Open AccessJournal ArticleDOI

Updating the American College of Rheumatology revised criteria for the classification of systemic lupus erythematosus.

Mph Marc C. Hochberg Md
- 01 Sep 1997 - 
- Vol. 40, Iss: 9, pp 1725-1725
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TLDR
In 1992, Piette and colleagues suggested that the ACR revised criteria be reevaluated in light of the above discoveries, and the presence and clinical associations or antiphospholipid antibodies in patients with SLE was suggested.
Abstract
In 1982, the Diagnostic and Therapeutic Criteria Committee of the American College of Rheumatology (ACR)published revised criteria for the classification of systemiclupus erythematosus (SLE) (1). During the ensuing decade several investigators, including Drs. Graham Hughes and Donato Alarcon-Segovia, among others, have described the presence and clinical associations or antiphospholipid antibodies in patients with SLE, as well as the occurrence of theprimary antiphospholipid syndrome (2-5). In 1992, Piette and colleagues suggested that the ACR revised criteria be reevaluated in light of the above discoveries (6).

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Citations
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Quality of life over time in patients with systemic lupus erythematosus

TL;DR: The SF-36 in SLE patients with established disease changed little over an 8-year period and changes in theSF-36 were not affected by disease activity, steroids, or damage accumulation during the interval, but were affected by the presence of fibromyalgia.
Journal ArticleDOI

Review of Classification Criteria for Systemic Lupus Erythematosus

TL;DR: The American College of Rheumatology classification criteria were devised in 1982 and in 1997, the immunologic disorder criterion was revised by a committee (without validation) and one of the most important laboratory tests, hypocomplementemia, was excluded entirely.
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Disturbed Th1, Th2, Th17 and T(reg) balance in patients with systemic lupus erythematosus

TL;DR: Even in quiescent SLE-patients T-cell homeostasis is aberrant in terms of skewing towards IL-17 producing T-cells, and the ability of T(regs) to express IFN-γ and IL17A was impaired in SLE -patients.
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The potential role of Th17 cells and Th17-related cytokines in the pathogenesis of lupus nephritis.

TL;DR: The results suggest the potential role of the IL-23/Th17 axis in the intra-renal inflammation of SLE patients and positively correlated with renal SLEDAI and histological activity index for LN patients.
BookDOI

Fundamentals of Inflammation

TL;DR: The text is presented as an introductory springboard for graduate students, postdoctoral Fellows, medical scientists, and researchers from other disciplines who wish to gain an appreciation and working knowledge of current cellular and molecular mechanisms fundamental to inflammation.
References
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Journal ArticleDOI

The 1982 revised criteria for the classification of systemic lupus erythematosus

TL;DR: The 1971 preliminary criteria for the classification of systemic lupus erythematosus (SLE) were revised and updated to incorporate new immunologic knowledge and improve disease classification and showed gains in sensitivity and specificity.
Journal ArticleDOI

Anticardiolipin antibodies: detection by radioimmunoassay and association with thrombosis in systemic lupus erythematosus

TL;DR: A new solid-phase radioimmunoassay for the detection of anticardiolipin antibodies is 200-400 times more sensitive than the precipitation method used in the Venereal Disease Reference Laboratory test and appears to have predictive value for thrombosis in SLE and related disorders.
Journal ArticleDOI

The "primary" antiphospholipid syndrome: major clinical and serological features.

TL;DR: The group of patients presented appears to be closely related, but distinctly separate from SLE, with a history of deep vein thromboses and a family history of SLE or a familial clotting tendency in a minority.
Journal Article

Evaluation of the anti-cardiolipin antibody test: report of an international workshop held 4 April 1986.

TL;DR: This study shows that properly performed ELISA or SRIA assays can be used to provide an accurate, reproducible, and quantitative measure of IgG and IgM aCL concentration in serum samples.
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