M
Michael A. Matthay
Researcher at University of California, San Francisco
Publications - 1063
Citations - 110857
Michael A. Matthay is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: Lung injury & Lung. The author has an hindex of 151, co-authored 998 publications receiving 98687 citations. Previous affiliations of Michael A. Matthay include University of California & Cardiovascular Institute of the South.
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Journal ArticleDOI
Therapeutic Effects of High Molecular Weight Hyaluronic Acid in Severe Pseudomonas Aeruginosa Pneumonia in Ex Vivo Perfused Human Lungs
Xiwen Zhang,Xiwen Zhang,Shinji Sugita,Shinji Sugita,Airan Liu,Yoshifumi Naito,Wonjung Hwang,Haibo Qiu,Atsuhiro Sakamoto,Teiji Sawa,Michael A. Matthay,Jae-Woo Lee +11 more
TL;DR: In this paper, extracellular vesicles (EVs) released during Escherichia coli (E. coli) bacterial pneumonia were inflammatory, and administration of high molecular weight hyaluronic aci...
Journal Article
American Thoracic Society Meeting 2016.: Mitochondrial Transfer Via Tunnelling Nanotubes (TNT) Is a Novel Mechanism by Which Mesenchymal Stromal Cells Enhance Macrophage Phagocytosis in In Vivo Models of Acute Lung Injury
Megan Jackson,Thomas Morrison,Daniel F. McAuley,Michael A. Matthay,Cecilia O'Kane,Anna Krasnodembskaya +5 more
Journal ArticleDOI
Exposure of mesenchymal stem cells to DMSO post-thaw affects cell viability and signaling
Stuart L. Gibb,Stuart L. Gibb,Michael A. Matthay,Frank Nizzi,Melissa Marlowe,Brian Jones,Shibani Pati,Shibani Pati +7 more
Posted ContentDOI
Immediate myeloid depot for SARS-CoV-2 in the human lung
Mark Einer Looney,Mélia Magnen,Ran You,Arjun Rao,Ryan Davis,Lauren Rodriguez,Camille R. Simoneau,Lisiena Hysenaj,Kenneth H. Hu,C. Love,Prescott G. Woodruff,David J. Erle,Carolyn M. Hendrickson,Carolyn S. Calfee,Michael A. Matthay,Jeroen Roose,Anita Sil,Melanie Ott,Charles Langelier,Matthew E Krummel +19 more
TL;DR: In the early phase of SARS-CoV-2 infection, myeloid cells may provide a safe harbor for the virus with minimal immune stimulatory cues being generated, resulting in effective viral colonization and quenching of the immune system.