M
Michael A. Matthay
Researcher at University of California, San Francisco
Publications - 1063
Citations - 110857
Michael A. Matthay is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: Lung injury & Lung. The author has an hindex of 151, co-authored 998 publications receiving 98687 citations. Previous affiliations of Michael A. Matthay include University of California & Cardiovascular Institute of the South.
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Journal ArticleDOI
Health-related quality of life after acute lung injury.
Posted ContentDOI
Global Absence and Targeting of Protective Immune States in Severe COVID-19.
Alexis J. Combes,Tristan Courau,Nicholas F. Kuhn,Kenneth H. Hu,Arja Ray,William S. Chen,Simon J. Cleary,Nayvin W. Chew,Divyashree Kushnoor,Gabriella C Reeder,Alan Shen,Jessica Tsui,Kamir J Hiam-Galvez,Priscila Muñoz-Sandoval,Wandi S Zhu,David Lee,Yang Sun,Ran You,Mélia Magnen,Lauren Rodriguez,Aleksandra Leligdowicz,Colin R. Zamecnik,Rita P. Loudermilk,Michael R. Wilson,Chun Jimmie Ye,Gabriela K. Fragiadakis,Mark R. Looney,Vincent Chan,Alyssa Ward,Sidney A. Carrillo,Michael A. Matthay,David J. Erle,Prescott G. Woodruff,Charles Langelier,Kirsten N. Kangelaris,Carolyn M. Hendrickson,Carolyn S. Calfee,Arjun A. Rao,Matthew F. Krummel +38 more
TL;DR: Overzealous and auto-directed antibody responses pit the immune system against itself in many COVID-19 patients and this defines targets for immunotherapies to allow immune systems to provide viral defense.
Journal ArticleDOI
Extracellular Vesicles: A New Frontier for Research in Acute Respiratory Distress Syndrome.
Rahul Y Mahida,Rahul Y Mahida,Shotaro Matsumoto,Shotaro Matsumoto,Michael A. Matthay,Michael A. Matthay +5 more
TL;DR: The important role EVs play in both inducing and attenuating inflammatory lung injury in ARDS as well as in sepsis, the most important clinical cause of ARDS is discussed.
Journal ArticleDOI
Cardiovascular-pulmonary monitoring in the intensive care unit. (Part 2)
TL;DR: Pitfalls in the acquisition and interpretation of data must be recognized before appropriate therapeutic decisions can be made and advanced monitoring devices and techniques must supplement and not supplant clinical assessment.
Journal ArticleDOI
Regulated gene expression in cultured type II cells of adult human lung
Philip L. Ballard,Jae Won Lee,Xiaohui Fang,Cheryl J. Chapin,Lennell Allen,Mark R. Segal,Horst Fischer,Beate Illek,Linda W. Gonzales,Venkatadri Kolla,Michael A. Matthay +10 more
TL;DR: The results further define the adult human type II cell molecular phenotype and demonstrate that a subset of genes remains hormone responsive in cultured adult cells.