R
Robert S. Fulton
Researcher at Washington University in St. Louis
Publications - 254
Citations - 167513
Robert S. Fulton is an academic researcher from Washington University in St. Louis. The author has contributed to research in topics: Genome & Gene. The author has an hindex of 109, co-authored 230 publications receiving 143530 citations. Previous affiliations of Robert S. Fulton include University of Washington & Brown University.
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Single Cell T Cell Receptor Repertoire Profiling for Dogs
Zachary L. Skidmore,Hans Rindt,Shirley Chu,Bryan Fisk,Catrina Fronick,Robert S. Fulton,Mingyi Zhou,Nathan J. Bivens,Carol Norris Reinero,Malachi Griffith,Jeffrey N. Bryan,Obi L. Griffith +11 more
TL;DR: The developed reagents successfully generated scTCRseq data, for the first time, which allowed the T cell repertoire to be surveyed in dogs responding to anti-tumor immunotherapy trials, and will allow longitudinal tracking of anti-Tumor T cell dynamics in canine cancer immunotherapy Trials.
Journal ArticleDOI
Complete Sequencing and Comparison of 12 Normal Karyotype M1 AML Genomes with 12 t(15;17) Positive M3-APL Genomes
John S. Welch,David E. Larson,Li Ding,Michael D. McLellan,Tamara Lamprecht,Cyriac Kandoth,Jacqueline E. Payton,Jack Baty,Chris Harris,Cheryl F. Lichti,Robert S. Fulton,David J. Dooling,Daniel C. Koboldt,Heather Schmidt,Qunyuan Zhang,John R. Osborne,Ling Lin,Michelle O'Laughlin,Joshua F. McMichael,Kim D. Delehaunty,Sean McGrath,Lucinda Fulton,Vincent Magrini,Tammi L. Vickery,Todd Wylie,Jason Walker,Peter Westervelt,Michael H. Tomasson,Mark A. Watson,Sharon Heath,William D. Shannon,Rakesh Nagarajan,Daniel C. Link,Timothy A. Graubert,John F. DiPersio,Elaine R. Mardis,Richard K. Wilson,Timothy J. Ley +37 more
TL;DR: To characterize the genomic events associated with distinct subtypes of AML, whole genome sequencing was used to compare 24 tumor/normal sample pairs from patients with normal karyotype M1-AML and t(15;17)-positive M3-AMl to identify a new leukemic pathway and several important themes emerged.
Journal ArticleDOI
Recurrent switch 2 domain RAC2 mutations in intravascular large B-cell lymphoma
Rohan Kodgule,Pooja Khonde,Joshua Robinson,Amy D'Albora,Lisa Fineberg Cook,Catrina Fronick,Robert S. Fulton,Sridhar Nonavinkere Srivatsan,Patrick J. Cimino,Eric J. Duncavage +9 more
TL;DR: In this article , the authors propose a solution to solve the problem of the problem: this article ] of "uniformity" and "uncertainty" of the solution.
Journal ArticleDOI
Convergent Clonal Evolution of Signaling Gene Mutations Is a Hallmark of Myelodysplastic Syndrome Progression.
Andrew J. Menssen,Ajay Khanna,Christopher A. Miller,Sridhar Nonavinkere Srivatsan,Gue Su Chang,Jin Shao,Joshua Robinson,Michele O'Laughlin,Catrina Fronick,Robert S. Fulton,Kimberly J Brendel,Sharon Heath,Raya Saba,John S. Welch,David H. Spencer,Jacqueline E. Payton,Peter Westervelt,John F. DiPersio,Daniel C. Link,Matthew J. Schuelke,Meagan A. Jacoby,Eric J. Duncavage,Timothy J. Key,Matthew J. Walter +23 more
TL;DR: Multivariate analysis revealed that MDS patients who had a signaling gene mutation had a higher risk of AML progression, potentially providing a biomarker for progression.
Proceedings ArticleDOI
Abstract PR03: The genomic landscape of diffuse intrinsic pontine glioma and pediatric non-brainstem high-grade glioma
Alexander K. Diaz,Gang Wu,Barbara S. Paugh,Yongjin Li,Xiaoyan Zhu,Sherri Rankin,Chunxu Qu,Xiang Chen,Junyuan Zhang,John Easton,Michael N. Edmonson,Charles Lu,Panduka Nagahawatte,Erin Hedlund,Michael Rusch,Stanley Pounds,Tong Lin,Arzu Onar-Thomas,Robert Huether,Richard W. Kriwacki,Matthew Parker,Pankaj Gupta,Jared Becksfort,Lei Wei,Heather L. Mulder,Kristy Boggs,Bhavin Vadodaria,Donald Yergeau,Kerri Ochoa,Robert S. Fulton,Lucinda S. Fulton,Chris Jones,Alberto Broniscer,Cynthia Wetmore,Amar Gajjar,Li Ding,Elaine R. Mardis,Richard K. Wilson,James R. Downing,David W. Ellison,Jinghui Zhang,Suzanne J. Baker +41 more
TL;DR: From these findings, novel tumor models to better understand pediatric high-grade glioma are established and new insight into the genetic events driving pediatric HGG tumorigenesis is provided.