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Robert S. Fulton

Researcher at Washington University in St. Louis

Publications -  254
Citations -  167513

Robert S. Fulton is an academic researcher from Washington University in St. Louis. The author has contributed to research in topics: Genome & Gene. The author has an hindex of 109, co-authored 230 publications receiving 143530 citations. Previous affiliations of Robert S. Fulton include University of Washington & Brown University.

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Evaluation of 16s rDNA-based community profiling for human microbiome research

TL;DR: The data production protocols used for this work are those used by the participating centers to produce 16S rDNA sequence for the Human Microbiome Project, and these results can be informative for interpreting the large body of clinical 16s rDNA data produced for this project.
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High-Dimensional Analysis Delineates Myeloid and Lymphoid Compartment Remodeling during Successful Immune-Checkpoint Cancer Therapy

TL;DR: Differences across all hematopoietic cells from syngeneic mouse tumors during unrestrained tumor growth or effective ICT are defined to support the hypothesis that this macrophage polarization/activation results from effects on circulatory monocytes and early macrophages entering tumors, rather than on pre-polarized mature intratumoral macrophaging.
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The Human Tumor Atlas Network: Charting Tumor Transitions Across Space and Time at Single-Cell Resolution

Orit Rozenblatt-Rosen, +373 more
- 16 Apr 2020 - 
TL;DR: The Human Tumor Atlas Network (HTAN), part of the NCI Cancer Moonshot Initiative, will establish a clinical, experimental, computational, and organizational framework to generate informative and accessible three-dimensional atlases of cancer transitions for a diverse set of tumor types.
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Immune Escape of Relapsed AML Cells after Allogeneic Transplantation

TL;DR: AML relapse after transplantation was not associated with the acquisition of previously unknown AML‐specific mutations or structural variations in immune‐related genes, but it was associated with dysregulation of pathways that may influence immune function, including down‐regulation of MHC class II genes, which are involved in antigen presentation.