S
Serpil C. Erzurum
Researcher at Cleveland Clinic Lerner Research Institute
Publications - 406
Citations - 34734
Serpil C. Erzurum is an academic researcher from Cleveland Clinic Lerner Research Institute. The author has contributed to research in topics: Asthma & Medicine. The author has an hindex of 87, co-authored 353 publications receiving 29654 citations. Previous affiliations of Serpil C. Erzurum include Pulmonary Vascular Research Institute & National Institutes of Health.
Papers
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Proceedings ArticleDOI
Somatic Deletion Of SMAD9 In Congenital Heart Disease-Associated Pulmonary Arterial Hypertension Leads To Altered MicroRNA Processing And Cell Hyperproliferation
Journal ArticleDOI
Genetic analyses of chr11p15.5 region identify MUC5AC-MUC5B associated with asthma-related phenotypes.
Xingnan Li,Huashi Li,Stephanie A. Christenson,Mario Castro,Loren C. Denlinger,Serpil C. Erzurum,John V. Fahy,Benjamin Gaston,Elliot Israel,Nizar N. Jarjour,Bruce D. Levy,David T. Mauger,Wendy C. Moore,Joe Zein,Naftali Kaminski,Sally E. Wenzel,Prescott G. Woodruff,Eugene R. Bleecker,Deborah A. Meyers +18 more
TL;DR: In this paper , the authors identified functional genes for asthma by combining SNPs and mRNA expression in bronchial epithelial cells (BEC) in the Severe Asthma Research Program (SARP).
Proceedings ArticleDOI
A Ferroptosis-Related Gene Signature Predicts Exacerbation-Prone Asthma
S. Xue,Jing Zhao,Seyed Mehdi Nouraie,S. Christenson,Michael C. Peters,John V. Fahy,P.G. Woodruff,Eugene R. Bleecker,Deborah A. Meyers,Loren C. Denlinger,Nizar N. Jarjour,Mario Castro,E. Israel,Bruce D. Levy,Serpil C. Erzurum,B.M. Gaston,Anuradha Ray,Valerian E. Kagan,Sally E. Wenzel +18 more
Journal ArticleDOI
Nitric oxide alters hyaluronan deposition by airway smooth muscle cells
Alana K. Majors,Ritu Chakravarti,Lisa Ruple,Rachel Leahy,Dennis J. Stuehr,Mark E. Lauer,Serpil C. Erzurum,Serpil C. Erzurum,Allison J. Janocha,Mark A. Aronica,Mark A. Aronica +10 more
TL;DR: NO-induced production of leukocyte-binding HA by SMCs provides a new potential mechanism for the non-resolving airway inflammation in asthma and suggests a key role of non-immune cells in driving the chronic inflammation of the submucosa.