S
Serpil C. Erzurum
Researcher at Cleveland Clinic Lerner Research Institute
Publications - 406
Citations - 34734
Serpil C. Erzurum is an academic researcher from Cleveland Clinic Lerner Research Institute. The author has contributed to research in topics: Asthma & Medicine. The author has an hindex of 87, co-authored 353 publications receiving 29654 citations. Previous affiliations of Serpil C. Erzurum include Pulmonary Vascular Research Institute & National Institutes of Health.
Papers
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Low levels of nitric oxide and carbon monoxide in 1 -antitrypsin deficiency
Roberto Machado,James K Stoller,Daniel Laskowski,Shuo Zheng,Joseph A. Lupica,Raed A. Dweik,Serpil C. Erzurum +6 more
TL;DR: Demonstration of lower than normal CO and NO levels may be useful as an additional noninvasive method to evaluate 1-AT deficiency in individuals with a severe, early onset of obstructive lung disease.
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Low levels of nitric oxide and carbon monoxide in α1-antitrypsin deficiency
Roberto F. Machado,James K. Stoller,Daniel Laskowski,Shuo Zheng,Joseph A. Lupica,Raed A. Dweik,Serpil C. Erzurum +6 more
TL;DR: In this article, the authors hypothesized that exhaled CO is increased in individuals with high blood cholesterol levels, while NO and carbon monoxide levels were not increased in those with high cholesterol levels.
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The Impact of Insulin Resistance on Loss of Lung Function and Response to Treatment in Asthma.
Michael C. Peters,Mark L. Schiebler,Juan Carlos Cardet,Mats W. Johansson,Ronald L. Sorkness,Mark D. DeBoer,Eugene R. Bleecker,Deborah A. Meyers,Mario Castro,Kaharu Sumino,Serpil C. Erzurum,Matthew C. Tattersall,Joe Zein,Annette T. Hastie,Wendy C. Moore,Bruce D. Levy,Elliot Israel,Melody G. Duvall,Brenda R. Phillips,David T. Mauger,Sally E. Wenzel,Merritt L. Fajt,Suneil K. Koliwad,Loren C. Denlinger,Prescott G. Woodruff,Nizar N. Jarjour,John V. Fahy +26 more
TL;DR: Homeostatic measure of IR, lung function, and treatment responses to bronchodilator and corticosteroid treatments and those with IR had significantly lower values for forced expired volume in one second and forced vital capacity, and these lower values were not attributable to obesity effects.
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Mechanisms of right heart disease in pulmonary hypertension (2017 Grover Conference Series).
TL;DR: Current dogma is that pathological hypertrophy of the right ventricle is a direct consequence of pulmonary vascular remodeling, but the causes of maladaptive cardiac remodeling remain unknown and require further research.
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Impaired right ventricular-pulmonary vascular function in myeloproliferative neoplasms.
Emir Charles Roach,Margaret M. Park,W.H. Wilson Tang,James D. Thomas,Kewal Asosingh,Matt Kalaycio,Serpil C. Erzurum,Samar Farha,Samar Farha +8 more
TL;DR: An important role for myeloid progenitors in the maintenance of pulmonary-vascular health is suggested, in which abnormal myeloproliferative progenitor cells are associated with RV pathology.