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Depression increases the risk of hypertension incidence: a meta-analysis of prospective cohort studies.

TLDR
It is found that depression increased the risk of hypertension incidence and the risk was significantly correlated with the length of follow-up and the prevalence of depression at baseline, suggesting that depression is probably an independent risk factor of hypertension.
Abstract
Background:It has long been known that depression is associated with hypertension but whether depression is a risk factor for hypertension incidence is still inconclusive.Objectives:To assess whether depression increases the incidence of hypertension.Method:Literatures were searched from PubMed, EMB

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The Lancet Psychiatry Commission: a blueprint for protecting physical health in people with mental illness

Joseph Firth, +56 more
TL;DR: This Commission summarises advances in understanding on the topic of physical health in people with mental illness, and presents clear directions for health promotion, clinical care, and future research.
Journal ArticleDOI

State of the Art Review: Depression, Stress, Anxiety, and Cardiovascular Disease

TL;DR: This article reviews studies connecting depression, stress/PTSD, and anxiety to CVD, focusing on findings from the last 5 years and examines the epidemiologic evidence establishing a link with CVD.
Journal ArticleDOI

Understanding the somatic consequences of depression: biological mechanisms and the role of depression symptom profile

TL;DR: The heterogeneity of the depression concept seems to play a differentiating role: metabolic syndrome and inflammation up-regulations appear more specific to the atypical depression subtype, whereas hypercortisolemia appears more specific for melancholic depression.

Genetic variants associated with subjective well-being, depressive symptoms, and neuroticism identified through genome-wide analyses

Aysu Okbay, +179 more
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Depression and cardiovascular disease: Epidemiological evidence on their linking mechanisms.

TL;DR: People with MDD have significantly worse lifestyles as well as more pathophysiological disturbances as compared to healthy controls, and some of these differences seem to be specific for (typical versus ‘atypical’, or antidepressant treated versus drug‐naive) subgroups of MDD patients.
References
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Journal ArticleDOI

Preferred reporting items for systematic reviews and meta-analyses: the PRISMA statement

TL;DR: Moher et al. as mentioned in this paper introduce PRISMA, an update of the QUOROM guidelines for reporting systematic reviews and meta-analyses, which is used in this paper.
Journal ArticleDOI

Preferred reporting items for systematic reviews and meta-analyses: The PRISMA statement

TL;DR: A structured summary is provided including, as applicable, background, objectives, data sources, study eligibility criteria, participants, interventions, study appraisal and synthesis methods, results, limitations, conclusions and implications of key findings.

The Newcastle-Ottawa Scale (NOS) for Assessing the Quality of Nonrandomised Studies in Meta-Analyses

TL;DR: The Newcastle-Ottawa Scale (NOS) as discussed by the authors was developed to assess the quality of nonrandomised studies with its design, content and ease of use directed to the task of incorporating the quality assessments in the interpretation of meta-analytic results.
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The PRISMA statement for reporting systematic reviews and meta-analyses of studies that evaluate healthcare interventions: explanation and elaboration

TL;DR: The meaning and rationale for each checklist item is explained, and an example of good reporting is included and, where possible, references to relevant empirical studies and methodological literature are included.
Journal ArticleDOI

Protective and Damaging Effects of Stress Mediators

TL;DR: The long-term effect of the physiologic response to stress is reviewed, which I refer to as allostatic load, which is the ability to achieve stability through change.
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