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Petra H.M. Peeters

Researcher at Utrecht University

Publications -  720
Citations -  73551

Petra H.M. Peeters is an academic researcher from Utrecht University. The author has contributed to research in topics: European Prospective Investigation into Cancer and Nutrition & Breast cancer. The author has an hindex of 119, co-authored 720 publications receiving 63681 citations. Previous affiliations of Petra H.M. Peeters include Memorial Sloan Kettering Cancer Center & Medical Research Council.

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Vitamin D Receptor and Calcium Sensing Receptor Polymorphisms and the Risk of Colorectal Cancer in European Populations

TL;DR: A role for the BsmI polymorphism of the VDR gene in CRC risk is confirmed, independent of serum 25OHD concentration and dietary calcium intake.
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Evaluation of Overdiagnosis of Breast Cancer in Screening with Mammography: Results of the Nijmegen Programme

TL;DR: It is concluded that there is no evidence that screening programmes using modern mammography constitute a significant risk for overdiagnosis of breast cancers.
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NAT2 slow acetylation and GSTM1 null genotypes may increase postmenopausal breast cancer risk in long-term smoking women.

TL;DR: Support is provided for the view that women who smoke and who have a genetically determined reduced inactivation of carcinogens (GSTM1 null genotype or slow NAT2 genotype) are at increased risk of breast cancer.
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Plasma carotenoids, vitamin C, tocopherols, and retinol and the risk of breast cancer in the European Prospective Investigation into Cancer and Nutrition cohort

Marije F. Bakker, +51 more
TL;DR: It is indicated that higher concentrations of plasma β-carotene and α- carotene are associated with lower breast cancer risk of ER- tumors and no statistically significant interaction between smoking, alcohol, or BMI is observed.
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DNA repair polymorphisms and the risk of stomach adenocarcinoma and severe chronic gastritis in the EPIC-EURGAST study

TL;DR: The study supports a role of ERCC2 in non-cardial GC but not in cardial cancer, and is the first prospective study suggesting that individual variation in DNA repair may be relevant for gastric carcinogenesis.