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Yutaka Suzuki

Researcher at University of Tokyo

Publications -  875
Citations -  42146

Yutaka Suzuki is an academic researcher from University of Tokyo. The author has contributed to research in topics: Gene & Medicine. The author has an hindex of 85, co-authored 767 publications receiving 35471 citations. Previous affiliations of Yutaka Suzuki include Japan Agency for Medical Research and Development & Institute for the Physics and Mathematics of the Universe.

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Journal ArticleDOI

Predicting promoter activities of primary human DNA sequences

TL;DR: It is found that nucleosomes occasionally formed open chromatin structures with RNA polymerase II recruitment where the program predicted significant promoter activities, although no transcripts were observed, and the prediction accuracy of the program was evaluated using massive sequencing analysis of transcriptional start sites in vivo.
Patent

Electromechanical transducer and fluid control assembly equipped therewith

TL;DR: In this paper, an electromechanical transducer including a high precision and high responsive movable member and free from lowering in durability caused by a working fluid to be used is presented.
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Temperature dependence of switching field distribution in a NiFe wire with a pad

TL;DR: In this paper, the distribution of switching fields in a NiFe wire was investigated as a function of temperature between 5 and 300 K. The authors obtained a histogram of H sw with three narrow peaks at each temperature.
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RAB5A and TRAPPC6B are novel targets for Shiga toxin 2a inactivation in kidney epithelial cells.

TL;DR: Using RNAi to silence factors involved in ACHN cells’ Stx response, the greatest protection occurred when silencing RAB5A and TRAPPC6B, two host factors that are newly link to Stx trafficking and fully prevented the cytotoxic Stx effect.
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Remote solid cancers rewire hepatic nitrogen metabolism via host nicotinamide-N-methyltransferase

TL;DR: In this paper , it was shown that 1-methylnicotinamide-N-methyltransferase (NNMT) is a host factor that mediates metabolic dysfunction in the livers of cancer-bearing mice.