scispace - formally typeset
Y

Yutaka Suzuki

Researcher at University of Tokyo

Publications -  875
Citations -  42146

Yutaka Suzuki is an academic researcher from University of Tokyo. The author has contributed to research in topics: Gene & Medicine. The author has an hindex of 85, co-authored 767 publications receiving 35471 citations. Previous affiliations of Yutaka Suzuki include Japan Agency for Medical Research and Development & Institute for the Physics and Mathematics of the Universe.

Papers
More filters
Journal ArticleDOI

CLOCK-Controlled Polyphonic Regulation of Circadian Rhythms through Canonical and Noncanonical E-Boxes

TL;DR: 7,978 CLOCK-binding sites in mouse liver are identified by chromatin immunoprecipitation-sequencing (ChIP-Seq), and a newly developed bioinformatics method, motif centrality analysis of ChIP- Seq (MOCCS), revealed a genome-wide distribution of previously unappreciated noncanonical E-boxes targeted by CLOCK.
Journal ArticleDOI

Light Controls Protein Localization through Phytochrome-Mediated Alternative Promoter Selection

TL;DR: This study found that the plant photoreceptor phytochrome induces genome-wide changes in alternative promoter selection in Arabidopsis thaliana, and suggests that alternative promoter usage represents another ubiquitous layer of gene expression regulation in eukaryotes that contributes to diversification of the proteome.
Journal ArticleDOI

Diversity of Translation Start Sites May Define Increased Complexity of the Human Short ORFeome

TL;DR: A novel post-transcriptional system that can augment the human proteome via the alternative use of diverse translation start sites coupled with transcriptional regulation through alternative promoters or splicing, leading to increased complexity of short protein-coding regions defined by the human transcriptome is revealed.
Journal ArticleDOI

A Core Promoter and a Frequent Single-Nucleotide Polymorphism of the Mismatch Repair GenehMLH1

TL;DR: This research characterized the promoter region of the hMLH1 gene and searched for mutations correlating to HNPCC and found a single-nucleotide polymorphism at position -93 nt from the adenine residue of the start codon that is a suitable marker for the detection of h MLH1 allelic losses.