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David W. Johnson

Researcher at University of Queensland

Publications -  2880
Citations -  157072

David W. Johnson is an academic researcher from University of Queensland. The author has contributed to research in topics: Peritoneal dialysis & Kidney disease. The author has an hindex of 160, co-authored 2714 publications receiving 140778 citations. Previous affiliations of David W. Johnson include Minnesota Department of Transportation & Open University.

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Outcomes of transplants from patients with small renal tumours, live unrelated donors and dialysis wait‐listed patients

TL;DR: Transplantation of tumourectomized kidneys from patients with small, localized, incidentally detected renal tumours results in similar outcomes to conventional LURTs and confers a significant survival advantage for patients who would otherwise be unable to receive a transplant.
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Lattice Dynamics in the FeSb 3 Skutterudite

TL;DR: In this paper, a systematic comparison of the lattice dynamics with CoSb, EuFe and EuSb was conducted, and it was shown that the Fe${}{4}$Sb${}_{12}$ framework is softer than the Co{}-Sb{}{3}$ Sb{12} framework, and that the observed softening and the associated lowering of lattice thermal conductivity in the $R$Fe{}_{4}µSbµ filled skutterudites are not only related to
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Recommendations for the use of icodextrin in peritoneal dialysis patients

TL;DR: Patients with impaired ultrafiltration, particularly in the settings of acute peritonitis, high transporter status and diabetes mellitus, appear to derive the greatest benefit from icodextrin with respect to augmentation of dialytic fluid removal, amelioration of symptomatic fluid retention and possible prolongation of technique survival.
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In vitro activation of rat brain protein kinase C by polyenoic very-long-chain fatty acids.

TL;DR: A variety of fatty acids including the cis‐polyunsaturated very‐long‐chain fatty acids (VLCFA) common in retina, spermatozoa, and brain were examined for their ability to activate protein kinase C (PKC) purified from rat brain.
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Erythropoietin administration protects against functional impairment and cell death after ischaemic renal injury in pigs.

TL;DR: To determine whether the administration of erythropoietin at the time of ischaemic renal injury (IRI) inhibits apoptosis, enhances tubular epithelial regeneration and promotes renal functional recovery, as it does in rodent models, in a higher mammalian model.