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Josep M. Antó

Researcher at Pompeu Fabra University

Publications -  523
Citations -  45951

Josep M. Antó is an academic researcher from Pompeu Fabra University. The author has contributed to research in topics: Asthma & Population. The author has an hindex of 100, co-authored 493 publications receiving 38663 citations. Previous affiliations of Josep M. Antó include University of Rochester & Centra.

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Measurement of general health status of non-oxygen-dependent chronic obstructive pulmonary disease patients

TL;DR: Results underscore the importance of measuring symptoms carefully when assessing patients, whose health status is substantially affected by the Chronic obstructive pulmonary disease, and suggest that it is relevant to assess sleep disturbances in these patients.
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MASK 2017: ARIA digitally-enabled, integrated, person-centred care for rhinitis and asthma multimorbidity using real-world-evidence

Jean Bousquet, +93 more
TL;DR: An overview of the methods used in MASK and the key results obtained to date include a novel phenotypic characterization of the patients, confirmation of the impact of allergic rhinitis on work productivity and treatment patterns in real life and the potential usefulness of MASK will be further explored.
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Prenatal Particulate Air Pollution and DNA Methylation in Newborns: An Epigenome-Wide Meta-Analysis

Olena Gruzieva, +55 more
TL;DR: Several differentially methylated CpGs and DMRs associated with prenatal PM exposure were identified in newborns, with annotation to genes previously implicated in lung-related outcomes.
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Prospective Study of Physical Activity and Risk of Asthma Exacerbations in Older Women

TL;DR: Regular physical activity was associated with reduced risk of exacerbations in women with asthma in this longitudinal study and decreased with increasing level of physical activity.
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Anti-tissue antibodies are related to lung function in chronic obstructive pulmonary disease.

TL;DR: The observed relationship between AT and lung function supports a role for autoimmunity in the pathogenesis of COPD.