scispace - formally typeset
L

László Szabados

Researcher at MTA Biological Research Centre

Publications -  169
Citations -  19011

László Szabados is an academic researcher from MTA Biological Research Centre. The author has contributed to research in topics: Cepheid variable & Arabidopsis. The author has an hindex of 48, co-authored 146 publications receiving 14652 citations. Previous affiliations of László Szabados include Max Planck Society & University of Barcelona.

Papers
More filters
Journal ArticleDOI

The Gaia mission

T. Prusti, +624 more
TL;DR: Gaia as discussed by the authors is a cornerstone mission in the science programme of the European Space Agency (ESA). The spacecraft construction was approved in 2006, following a study in which the original interferometric concept was changed to a direct-imaging approach.
Journal ArticleDOI

Proline: a multifunctional amino acid

TL;DR: The compartmentalization of proline biosynthesis, accumulation and degradation in the cytosol, chloroplast and mitochondria is discussed and the role of prolines in cellular homeostasis, including redox balance and energy status, is described.
Journal ArticleDOI

Gaia Data Release 2: Observational Hertzsprung-Russell diagrams

C. Babusiaux, +451 more
TL;DR: In this article, the power of the Gaia DR2 in studying many fine structures of the Hertzsprung-Russell diagram (HRD) was highlighted, depending in particular on stellar population selections.
Journal ArticleDOI

Gaia Data Release 2 - Observational Hertzsprung-Russell diagrams

C. Babusiaux, +451 more
TL;DR: Gaia Data Release 2 provides high-precision astrometry and three-band photometry for about 1.3 billion sources over the full sky as mentioned in this paper, which is unprecedented in both precision and coverage of the various Milky Way stellar populations and stellar evolutionary phases.
Journal ArticleDOI

Duplicated P5CS genes of Arabidopsis play distinct roles in stress regulation and developmental control of proline biosynthesis

TL;DR: The genetic characterization of p5cs insertion mutants is described, which indicates that P5CS1 is required for proline accumulation under osmotic stress, and that P4CS2 is insufficient for compensation of developmental defects caused by inactivation of P5 CS2.