S
Suzanne Oparil
Researcher at University of Alabama at Birmingham
Publications - 941
Citations - 122414
Suzanne Oparil is an academic researcher from University of Alabama at Birmingham. The author has contributed to research in topics: Blood pressure & Angiotensin II. The author has an hindex of 106, co-authored 885 publications receiving 113983 citations. Previous affiliations of Suzanne Oparil include Michigan State University & Oregon Health & Science University.
Papers
More filters
Journal ArticleDOI
Outcomes of Shared IRB Compared with Multiple Individual Site IRB Models in a Multisite Clinical Trial.
Samantha L. Martin,Phillip H. Allman,Lorraine Dugoff,Baha M. Sibai,Stephanie A. Lynch,Jennifer Ferrara,Kjersti Aagaard,Christina Zornes,Jennifer L. Wilson,Marie Gibson,Molly M Adams,Sherri Longo,Amy Staples,George R. Saade,Imene Beche,Ebony B. Carter,Michelle Y. Owens,Hyagriv N. Simhan,Heather A. Frey,Shama N. Khan,Anna Palatnik,Phyllis August,L. Irby,Tiffany Lee,Christine A Lee,Paula Schum,Rosalyn Chan-Akeley,Catera Duhon,Monica Rincon,Kelly S. Gibson,Samantha Wiegand,D G Eastham,Suzanne Oparil,Jeff M. Szychowski,Alan T.N. Tita +34 more
TL;DR: In this article , the authors compare processing times and other processing characteristics between sites using a single IRB and those using their own site IRBs in a multicenter clinical trial, and find that the shared IRB model for multicenter studies may be more efficient in terms of cumulative time and effort required to obtain approval of an IRB protocol.
Journal ArticleDOI
Intensive systolic blood pressure control and prevention of new onset atrial fibrillation in the SPRINT study: is the association really controversial?
TL;DR: An apparent controversy has arisen about whether intensive systolic blood pressure control prevented new onset atrial fibrillation in the Systolic Blood Pressure Intervention Trial (SPRIN...
Journal ArticleDOI
Blunted responsiveness of posterior hypothalamic norepinephrine to quinpirole in DOCA/NaCl hypertensive rats
TL;DR: The data suggest that the specific D2 agonist may effect its central pressor response by stimulating NE release from posterior hypothalamic area, a "pressor" region of hypothalamus, and that this D 2 agonist induced pressor mechanism may be blunted in DOCA/NaCl hypertension.