scispace - formally typeset
S

Suzanne Oparil

Researcher at University of Alabama at Birmingham

Publications -  941
Citations -  122414

Suzanne Oparil is an academic researcher from University of Alabama at Birmingham. The author has contributed to research in topics: Blood pressure & Angiotensin II. The author has an hindex of 106, co-authored 885 publications receiving 113983 citations. Previous affiliations of Suzanne Oparil include Michigan State University & Oregon Health & Science University.

Papers
More filters
Journal ArticleDOI

Association between chronic kidney disease and cancer mortality: A report from the ALLHAT.

TL;DR: In a large cohort of hypertensive patients, GFR < 45 mL/min/1.73 m2 was associated with a higher risk of cancer-related mortality, consistent across subgroups defined by race, gender, and diabetes.
Journal ArticleDOI

Masked Uncontrolled Hypertension Is Accompanied by Increased Out-of-Clinic Aldosterone Secretion

TL;DR: This study suggests that patients with MUCH likely have higher out-of-clinic sympathetic nervous system tone increases aldosterone secretion mediated by increased renin release that may contribute to their higher out of-Clinic BP.
Journal ArticleDOI

Moderate versus intensive treatment of hypertension with amlodipine/valsartan for patients uncontrolled on angiotensin receptor blocker monotherapy

TL;DR: Starting treatment with an intensive dose of amlodipine/valsartan provides significantly greater BP lowering versus moderate treatment in hypertensive patients unresponsive to ARB monotherapy.
Journal ArticleDOI

Secondary prevention of coronary heart disease in women: a call to action.

TL;DR: Data is analyzed from the HERS study to identify risk factors for myocardial infarction and CHD death and describe use of secondary prevention strategies by the participants and to identify Elevated lipoprotein(a) levels emerged as a novel risk factor only in the placebo group.
Journal ArticleDOI

Treating multiple-risk hypertensive populations.

TL;DR: Lowering BP to levels well below the traditional goal of 140/90 mm Hg is needed to slow the progression of renal dysfunction and prevent renal failure in hypertensive patients with renal disease, whether related to diabetes or to another etiology.