S
Suzanne Oparil
Researcher at University of Alabama at Birmingham
Publications - 941
Citations - 122414
Suzanne Oparil is an academic researcher from University of Alabama at Birmingham. The author has contributed to research in topics: Blood pressure & Angiotensin II. The author has an hindex of 106, co-authored 885 publications receiving 113983 citations. Previous affiliations of Suzanne Oparil include Michigan State University & Oregon Health & Science University.
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Journal ArticleDOI
Primary aldosteronism: a practical approach to diagnosis and treatment.
Roopal B. Thakkar,Suzanne Oparil +1 more
TL;DR: The plasma aldosterone concentration/plasma renin activity ratio is a simple screening test for PA that should be performed in all patients with refractory/severe hypertension, spontaneous or provoked (by diuretics) hypokalemia, or a requirement for excessive potassium supplementation to maintain normoksalemia.
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Baseline characteristics of African Americans in the Systolic Blood Pressure Intervention Trial
Carolyn H Still,Timothy E. Craven,Barry I. Freedman,Peter N. Van Buren,Kaycee M. Sink,Anthony A. Killeen,Jeffrey T. Bates,Alberta Bee,Gabriel Contreras,Suzanne Oparil,Carolyn M. Pedley,Barry M. Wall,Suzanne White,Delia M. Woods,Carlos J. Rodriguez,Jackson T. Wright +15 more
TL;DR: Multivariate logistic regression analysis found BP control rates to <140/90 mm Hg were higher for AAs who were male, had higher number of chronic diseases, were on diuretic treatment, and had better medication adherence.
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2-Tetradecylglycidic Acid, an Inhibitor of Carnitine Palmitoyltransferase-1, Induces Myocardial Hypertrophy via the AT1 Receptor
Paul E. Wolkowicz,Ferdinand Urthaler,Clarence Forrest,Hai Shen,Joan Durand,Chih-Chang Wei,Suzanne Oparil,Louis J. Dell′Italia +7 more
TL;DR: Treating rats with TDGA or giving rats drinking water that contained 1 mg losartan/ml altered neither their heart rate nor their mean arterial blood pressure when compared with untreated rats, suggesting that oxirane carboxylates induce myocardial hypertrophy by activating the AT1 receptor independent of changes in systemic hemodynamics.
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PTH, FGF23, and Intensive Blood Pressure Lowering in Chronic Kidney Disease Participants in SPRINT.
Charles Ginsberg,Charles Ginsberg,Timothy E. Craven,Michel Chonchol,Alfred K. Cheung,Mark J. Sarnak,Walter T. Ambrosius,Anthony A. Killeen,Kalani L. Raphael,Udayan Bhatt,Jing Chen,Glenn M. Chertow,Barry I. Freedman,Suzanne Oparil,Vasilios Papademetriou,Barry M. Wall,Clinton B. Wright,Joachim H. Ix,Joachim H. Ix,Michael G. Shlipak,Michael G. Shlipak +20 more
TL;DR: SPRINT participants with CKD and a high serum PTH received less cardiovascular protection from intensive BP therapy than participants with a lower serum P TH, and FGF23 was not independently associated with any outcome and did not modify the effects of the intervention.
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α2A-Adrenergic Receptors Mediate Sympathoinhibitory Responses to Atrial Natriuretic Peptide in the Mouse Anterior Hypothalamic Nucleus
TL;DR: It is demonstrated that in the anterior hypothalamic nucleus of the mouse (and probably in the rat) &agr;2A-adrenergic receptors mediate both sympathoinhibitory responses to &agR;2-ADrenergic receptor agonists and the action of atrial natriuretic peptide.