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Marc A. Pfeffer

Researcher at Brigham and Women's Hospital

Publications -  815
Citations -  143710

Marc A. Pfeffer is an academic researcher from Brigham and Women's Hospital. The author has contributed to research in topics: Heart failure & Myocardial infarction. The author has an hindex of 166, co-authored 765 publications receiving 133043 citations. Previous affiliations of Marc A. Pfeffer include Partners HealthCare & University of Miami.

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Lixisenatide in Type 2 Diabetes and Acute Coronary Syndrome

TL;DR: In analyses of death from cardiovascular causes, which was the principal component of the primary outcome affected by the intervention, there was no evidence of heterogeneity, and subgroup analyses revealed no heterogeneity in the effect of empagliflozin according to the use or nonuse of diuretic therapy at baseline.
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Challenges in conducting clinical trials in nephrology: conclusions from a Kidney Disease-Improving Global Outcomes (KDIGO) Controversies Conference.

Colin Baigent, +66 more
- 01 Aug 2017 - 
TL;DR: The main findings from the conference are summarized and a range of potential solutions are set out to ensure future trials among people with kidney disease are sufficiently robust to provide reliable answers and are not constrained by inappropriate complexities in design or conduct.
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Ventricular Remodeling Does Not Accompany the Development of Heart Failure in Diabetic Patients After Myocardial Infarction

TL;DR: Data from the Survival and Ventricular Enlargement (SAVE) echocardiographic substudy was used to test the hypothesis that diabetes was associated with increased LV enlargement after myocardial infarction.
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Left ventricular thrombus formation after first anterior wall acute myocardial infarction.

TL;DR: E ejection fraction, extent of early shape change and LV end-diastolic pressure as independent correlates of LV thrombus after acute myocardial infarction are identified.
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Increased left ventricular volume following myocardial infarction in man.

TL;DR: It is demonstrated that in the chronic phase of infarction,left ventricular remodeling results in alterations in left ventricular volumes that are proportional to the degree of wall motion abnormality.