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Institution

University of South Florida

EducationTampa, Florida, United States
About: University of South Florida is a education organization based out in Tampa, Florida, United States. It is known for research contribution in the topics: Population & Poison control. The organization has 34231 authors who have published 72644 publications receiving 2538044 citations. The organization is also known as: USF.


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Journal ArticleDOI
TL;DR: The findings from Study 1 indicated that level of obesity and perceived weight, but not maturational timing, had a directional influence on the other variables, and teasing history was significantly related to the development of body image and eating disturbance.
Abstract: Objective: Covariance structure modeling (CSM) and longitudinal analyses were used to identify a possible causal sequence for factors that might lead to the development of body image and eating disturbance in adolescent females. Method: In Study 1, subjects were measured for level of obesity, perceived weight status, maturational timing, history of being teased about weight/size, body image, eating disturbance, and global psychological functioning. Study 2 was a systematic replication of Study 1 using different indices of body image and a more comprehensive assessment of eating disturbance. In Study 3, subjects from Study 1 were reassessed at a 3-year follow-up. Results: The findings from Study 1 indicated that level of obesity and perceived weight, but not maturational timing, had a directional influence on the other variables. In addition, teasing history was significantly related to the development of body image and eating disturbance. CSM and path analyses with Studies 2 and 3 data generally replicated and extended the results from Study 1. Discussion: These findings offer important insights into possible causal sequences for the development of body image and eating disturbance. © 1995 by John Wiley & Sons, Inc.

406 citations

Journal ArticleDOI
TL;DR: The Good Behavior Game (GBG) was used in first and second-grade classrooms in 19 Baltimore City Public Schools beginning in the 1985-1986 school year as discussed by the authors, where the intervention was directed at the classroom as a whole to socialize children to the student role and reduce aggressive, disruptive behaviors.

406 citations

Journal ArticleDOI
TL;DR: In this article, the authors empirically tested a model to evaluate the impact of cause-brand alliances on subsequent attitudes toward both partners and found that the cause appears to benefit from the alliance to a greater extent than the brand.
Abstract: Partnering charitable causes with brands has become a common practice for many marketing programs; it is referred to strategically as cause-related marketing. Although there is the perception that both partners benefit from the alliance, research has focused primarily on the benefits to the brand. Using Attitude Accessibility, Congruity, and Information Integration Theories, this study empirically tests a model to evaluate the impact of cause–brand alliances on subsequent attitudes toward both partners. The results of the study (n = 463) support the assumption that attitudes toward both the cause and the brand can be enhanced as a consequence of an alliance if perceptions of the alliance are favorable. Furthermore, the cause appears to benefit from the alliance to a greater extent than the brand. The study supports the notions that the fit between partners plays a pivotal role in consumer acceptance of the alliance as plausible and that familiarity with the cause moderates the effectiveness of the alliance. These results represent a necessary step in developing a theoretical model to explain the effects of a cause–brand alliance on both partners. © 2004 Wiley Periodicals, Inc.

406 citations

Journal ArticleDOI
TL;DR: Strategies for reducing cancer risk in Hispanic populations include targeted, culturally appropriate interventions for increasing the uptake of preventive services and reduced cancer risk factor prevalence, as well as additional funding for Puerto Rico‐specific and subgroup‐specific cancer research and surveillance.
Abstract: Cancer is the leading cause of death among Hispanics/Latinos, who represent the largest racial/ethnic minority group in the United States, accounting for 17.8% (57.5 million) of the total population in the continental United States and Hawaii in 2016. In addition, more than 3 million Hispanic Americans live in the US territory of Puerto Rico. Every 3 years, the American Cancer Society reports on cancer occurrence, risk factors, and screening for Hispanics in the United States based on data from the National Cancer Institute, the North American Association of Central Cancer Registries, and the Centers for Disease Control and Prevention. For the first time, contemporary incidence and mortality rates for Puerto Rico, which has a 99% Hispanic population, are also presented. An estimated 149,100 new cancer cases and 42,700 cancer deaths will occur among Hispanics in the continental United States and Hawaii in 2018. For all cancers combined, Hispanics have 25% lower incidence and 30% lower mortality compared with non-Hispanic whites, although rates of infection-related cancers, such as liver, are up to twice as high in Hispanics. However, these aggregated data mask substantial heterogeneity within the Hispanic population because of variable cancer risk, as exemplified by the substantial differences in the cancer burden between island Puerto Ricans and other US Hispanics. For example, during 2011 to 2015, prostate cancer incidence rates in Puerto Rico (146.6 per 100,000) were 60% higher than those in other US Hispanics combined (91.6 per 100,000) and 44% higher than those in non-Hispanic whites (101.7 per 100,000). Prostate cancer is also the leading cause of cancer death among men in Puerto Rico, accounting for nearly 1 in 6 cancer deaths during 2011-2015, whereas lung cancer is the leading cause of cancer death among other US Hispanic men combined. Variations in cancer risk are driven by differences in exposure to cancer-causing infectious agents and behavioral risk factors as well as the prevalence of screening. Strategies for reducing cancer risk in Hispanic populations include targeted, culturally appropriate interventions for increasing the uptake of preventive services and reducing cancer risk factor prevalence, as well as additional funding for Puerto Rico-specific and subgroup-specific cancer research and surveillance.

406 citations

Journal ArticleDOI
Jerry S. Wolinsky1, Ponnada A. Narayana1, Paul O'Connor2, P. K. Coyle3, Corey C. Ford4, Kenneth P. Johnson5, Kenneth P. Johnson6, Aaron Miller6, Aaron Miller7, Lillian Pardo, Shaul Kadosh, David Ladkani, Lorne F. Kastrukoff8, Pierre Duquette9, Mark S. Freedman10, Marc Debouverie, Catherine Lubetski11, Gilles Edan, E Roullet, Christian Confavreux6, Alan J. Thompson, L D Blumhardt12, L D Blumhardt6, Stanley Hawkins, Thomas F. Scott13, Daniel Wynn, Joanna Cooper, Stephen Thurston, Stanton B. Elias14, Clyde E. Markowitz15, David Mattson16, John H. Noseworthy17, Elizabeth A. Shuster17, Jonathan L. Carter17, Fred D. Lublin18, WH Stuart19, Michael D. Kaufman, Gary Birnbaum, Kottil Rammohan20, Ruth H. Whitham21, Cornelia Mihai22, Steven J. Greenberg23, Craig M. Smith, Mark A. Agius24, Stan Van Den Noort25, Lawrence W. Myers26, James G. Nelson27, Douglas S. Goodin28, Barry G. W. Arnason29, Khurram Bashir30, Sharon G. Lynch31, Patricia K. Coyle3, Stephen Kamin32, William A. Sheremata33, Galen Mitchell34, Andrew D. Goodman35, Norman J Kachuck36, Peter B. Dunne37, J. William Lindsey1, Elliot M. Frohman38, James D. Bowen39, Benjamin Rix Brooks40, John W. Rose41, Harold L. Moses42, Douglas Jeffrey43, Anne H. Cross44, Robert P. Lisak45, Timothy Vollmer46, Jack P. Antel47, Gary Cutter, Luanne M. Metz48, Henry F. McFarland49, Steven Reingold, Fred D. Lublin6, Irina Vainrub, Lucie Lambert, Fengwei Zhong, Jeff Rasmituth, Saria Momin, Rivka Kreitman, Galia Shifroni, Irit Pinchasi, Yafit Stark 
University of Texas Health Science Center at Houston1, University of Toronto2, Stony Brook University3, University of New Mexico4, University of Maryland, Baltimore5, Icahn School of Medicine at Mount Sinai6, Maimonides Medical Center7, University of British Columbia8, Université de Montréal9, University of Ottawa10, University of Paris11, Queen's University12, Allegheny General Hospital13, Henry Ford Health System14, University of Pennsylvania15, Indiana University – Purdue University Indianapolis16, Mayo Clinic17, Drexel University18, Shepherd Center19, Ohio State University20, Oregon Health & Science University21, State University of New York Upstate Medical University22, Roswell Park Cancer Institute23, University of California, Davis24, University of California, Irvine25, University of California, Los Angeles26, University of California, San Diego27, University of California, San Francisco28, University of Chicago29, University of Alabama at Birmingham30, University of Kansas31, Rutgers University32, University of Miami33, University of Pittsburgh34, University of Rochester35, University of Southern California36, University of South Florida37, University of Texas Southwestern Medical Center38, University of Washington39, University of Wisconsin-Madison40, University of Utah41, Vanderbilt University42, Wake Forest University43, Washington University in St. Louis44, Wayne State University45, Yale University46, McGill University47, Foothills Medical Centre48, National Institutes of Health49
TL;DR: To determine whether glatiramer acetate slows accumulation of disability in primary progressive multiple sclerosis, a new drug is developed that acts as a ‘spatially aggregating agent’ to reduce the risk of disease progression.
Abstract: Objective To determine whether glatiramer acetate (GA) slows accumulation of disability in primary progressive multiple sclerosis. Methods A total of 943 patients with primary progressive multiple sclerosis were randomized to GA or placebo (PBO) in this 3-year, double-blind trial. The primary end point was an intention-to-treat analysis of time to 1- (entry expanded disability status scale, 3.0–5.0) or 0.5-point expanded disability status scale change (entry expanded disability status scale, 5.5–6.5) sustained for 3 months. The trial was stopped after an interim analysis by an independent data safety monitoring board indicated no discernible treatment effect on the primary outcome. Intention-to-treat analyses of disability and magnetic resonance imaging end points were performed. Results There was a nonsignificant delay in time to sustained accumulated disability in GA- versus PBO-treated patients (hazard ratio, 0.87 [95% confidence interval, 0.71–1.07]; p = 0.1753), with significant decreases in enhancing lesions in year 1 and smaller increases in T2 lesion volumes in years 2 and 3 versus PBO. Post hoc analysis showed that survival curves for GA-treated male patients diverged early from PBO-treated male subjects (hazard ratio, 0.71 [95% confidence interval, 0.53–0.95]; p = 0.0193). Interpretation The trial failed to demonstrate a treatment effect of GA on primary progressive multiple sclerosis. Both the unanticipated low event rate and premature discontinuation of study medication decreased the power to detect a treatment effect. Post hoc analysis suggests GA may have slowed clinical progression in male patients who showed more rapid progression when untreated. Ann Neurol 2007;61:14–24

406 citations


Authors

Showing all 34549 results

NameH-indexPapersCitations
David J. Hunter2131836207050
Aaron R. Folsom1811118134044
John Hardy1771178171694
David Cella1561258106402
Arul M. Chinnaiyan154723109538
Andrew D. Hamilton1511334105439
Charles B. Nemeroff14997990426
C. Ronald Kahn14452579809
Alexander Belyaev1421895100796
Tasuku Honjo14171288428
Weihong Tan14089267151
Alison Goate13672185846
Peter Kraft13582182116
Xiaodong Wang1351573117552
Lars Klareskog13169763281
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Performance
Metrics
No. of papers from the Institution in previous years
YearPapers
2023133
2022523
20214,289
20204,119
20193,710
20183,405