Devon Shook

TL;DR: Findings suggest an interplay in the abnormal development of the striatal, frontal, and temporal regions in ASD across the lifespan, using a well-established, validated, publicly available analysis pipeline.

TL;DR: It is suggested thatChildren with ASD+ADHD symptoms present with exacerbated impairments in some but not all domains of functioning relative to children with ASD, most notably in adaptive behavior and working memory.

TL;DR: Pediatric success rates with epilepsy, attention deficit/hyperactivity disorder (ADHD), autism spectrum disorders (ASD), and typically developing children (TYP) are examined and 20–30% more children should be recruited in these patient groups, but only 10–20% for TYP for research studies.

TL;DR: Altered lateralized neurodevelopment may be a feature of ASD, affecting widespread brain regions with diverse functions, and particularly in medial frontal, orbitofrontal, cingulate and inferior temporal areas.

TL;DR: In this article, the authors used T1-weighted magnetic resonance images to determine neurobiologic correlates of group differences in cortical thickness between cases and controls in 6 disorders: attention-deficit/hyperactivity disorder (ADHD), autism spectrum disorder (ASD), bipolar disorder (BD), major depressive disorder (MDD), obsessive-compulsive disorder (OCD), and schizophrenia.