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Jan Reedijk

Researcher at Leiden University

Publications -  1165
Citations -  45902

Jan Reedijk is an academic researcher from Leiden University. The author has contributed to research in topics: Ligand & Crystal structure. The author has an hindex of 80, co-authored 1163 publications receiving 43747 citations. Previous affiliations of Jan Reedijk include Nagoya City University & University of Münster.

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Synthesis, crystal structure, spectroscopic and magnetic properties of doubly and triply bridged dinuclear copper(II) compounds containing di-2-pyridylamine as a ligand

TL;DR: The dihydroxo-bridged dinuclear copper(II) compound [Cu2(dpyam)2(μ-OH)2]I2 (1) and the triply bridged Dinuclear copper (II) compounds with a formato bridge as discussed by the authors were synthesized and their crystal structures determined by X-ray crystallographic methods.
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A copper complex bearing a TEMPO moiety as catalyst for the aerobic oxidation of primary alcohols

TL;DR: A new bifunctional, triazine-based ligand has been designed with the aim to generate a copper(II) complex holding a TEMPO (2,2,6,6-tetramethylpiperidinyloxy) moiety, which is capable of catalysing the selective, aerobic oxidation of benzyl alcohol to 84% of benzaldehyde in 24 h.
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Sense and nonsense of science citation analyses: comments on the monopoly position of ISI and citation inaccuracies. Risks of possible misuse and biased citation and impact data.

TL;DR: In this article, a review of the current practices in publications and citations for bio-chemists and bio-chemist journals is presented, where critical comments are made with regard to the use and consequences of erroneous and incomplete or too detailed data.
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Synthesis, structure, spectroscopy, and magnetism of two new dinuclear carbonato-bridged Cu(II) complexes

TL;DR: In this paper, two dinuclear μ-CO3−2− Cu(II) complexes with different coordination modes for the carbonato bridge have been obtained by fixation of atmospheric CO2 and also directly prepared from the carbonate salt.
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High cytotoxicity of cisplatin nanocapsules in ovarian carcinoma cells depends on uptake by caveolae-mediated endocytosis.

TL;DR: The high cytotoxicity of cisPlatin nanocapsules requires caveolin-1-dependent endocytosis that is followed by release of the drug from a late endosomal/lysosomal compartment and cisplatin-DNA-adduct formation and the findings may be applied in predicting the efficacy of nanoparticulate anticancer drug delivery systems in treating different tumor types.