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Lynda Chin

Researcher at University of Texas MD Anderson Cancer Center

Publications -  127
Citations -  49829

Lynda Chin is an academic researcher from University of Texas MD Anderson Cancer Center. The author has contributed to research in topics: Melanoma & Cancer. The author has an hindex of 64, co-authored 126 publications receiving 40137 citations. Previous affiliations of Lynda Chin include Massachusetts Institute of Technology & Heidelberg University.

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Journal Article

The Cancer Genome Atlas Pan-Cancer analysis project

Kyle Chang, +337 more
- 01 Sep 2013 - 
TL;DR: The Cancer Genome Atlas (TCGA) Research Network has profiled and analyzed large numbers of human tumors to discover molecular aberrations at the DNA, RNA, protein and epigenetic levels as mentioned in this paper.
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Comprehensive molecular characterization of gastric adenocarcinoma

Adam J. Bass, +257 more
- 11 Sep 2014 - 
TL;DR: A comprehensive molecular evaluation of 295 primary gastric adenocarcinomas as part of The Cancer Genome Atlas (TCGA) project is described and a molecular classification dividing gastric cancer into four subtypes is proposed.
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Comprehensive molecular profiling of lung adenocarcinoma: The cancer genome atlas research network

Eric A. Collisson, +318 more
- 01 Jan 2014 - 
TL;DR: In this paper, the authors report molecular profiling of 230 resected lung adnocarcinomas using messenger RNA, microRNA and DNA sequencing integrated with copy number, methylation and proteomic analyses.
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The Somatic Genomic Landscape of Glioblastoma

Cameron Brennan, +57 more
- 10 Oct 2013 - 
TL;DR: Correlative analyses confirm that the survival advantage of the proneural subtype is conferred by the G-CIMP phenotype, and MGMT DNA methylation may be a predictive biomarker for treatment response only in classical subtype GBM.
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Comprehensive genomic characterization of head and neck squamous cell carcinomas

Michael S. Lawrence, +309 more
- 29 Jan 2015 - 
TL;DR: It is shown that human-papillomavirus-associated tumours are dominated by helical domain mutations of the oncogene PIK3CA, novel alterations involving loss of TRAF3, and amplification of the cell cycle gene E2F1.