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Reedik Mägi

Researcher at University of Tartu

Publications -  344
Citations -  57727

Reedik Mägi is an academic researcher from University of Tartu. The author has contributed to research in topics: Genome-wide association study & Population. The author has an hindex of 94, co-authored 306 publications receiving 47988 citations. Previous affiliations of Reedik Mägi include University of Oxford & Wellcome Trust Centre for Human Genetics.

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Genetic studies of body mass index yield new insights for obesity biology

Adam E. Locke, +481 more
TL;DR: This paper conducted a genome-wide association study and meta-analysis of body mass index (BMI), a measure commonly used to define obesity and assess adiposity, in up to 339,224 individuals.
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New genetic loci implicated in fasting glucose homeostasis and their impact on type 2 diabetes risk

Josée Dupuis, +339 more
- 01 Feb 2010 - 
TL;DR: It is demonstrated that genetic studies of glycemic traits can identify type 2 diabetes risk loci, as well as loci containing gene variants that are associated with a modest elevation in glucose levels but are not associated with overt diabetes.
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A genome-wide association search for type 2 diabetes genes in African Americans.

Nichole D. Palmer, +384 more
- 04 Jan 2012 - 
TL;DR: It is suggested that multiple loci underlie T2DM susceptibility in the African-American population and that these loci are distinct from those identified in other ethnic populations.

Association analyses of 249,796 individuals reveal 18 new loci associated with body mass index

Elizabeth K. Speliotes, +374 more
TL;DR: 18 new loci associated with body mass index are identified, one of which includes a copy number variant near GPRC5B, and genes in other newly associated loci may provide new insights into human body weight regulation.
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Defining the role of common variation in the genomic and biological architecture of adult human height

Andrew R. Wood, +444 more
- 01 Nov 2014 - 
TL;DR: This article identified 697 variants at genome-wide significance that together explained one-fifth of the heritability for adult height, and all common variants together captured 60% of heritability.