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Tomohiro Katsuya

Researcher at Osaka University

Publications -  236
Citations -  15108

Tomohiro Katsuya is an academic researcher from Osaka University. The author has contributed to research in topics: Population & Essential hypertension. The author has an hindex of 63, co-authored 222 publications receiving 13105 citations. Previous affiliations of Tomohiro Katsuya include Stanford University & University of Alabama at Birmingham.

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Novel genetic associations for blood pressure identified via gene-alcohol interaction in up to 570K individuals across multiple ancestries

Mary F. Feitosa, +299 more
- 18 Jun 2018 - 
TL;DR: In insights into the role of alcohol consumption in the genetic architecture of hypertension, a large two-stage investigation incorporating joint testing of main genetic effects and single nucleotide variant (SNV)-alcohol consumption interactions is conducted.
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Hypoadiponectinemia Is an Independent Risk Factor for Hypertension

TL;DR: Hypoadiponectinemia is a marker for predisposition to hypertension in men and blood pressure was inversely associated with adiponectin concentration in normotensives regardless of insulin resistance.
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Meta-analysis of genome-wide association studies identifies common variants associated with blood pressure variation in east Asians

TL;DR: A meta-analysis of genome-wide association studies of systolic (SBP) and diastolic (DBP) blood pressure in 19,608 subjects of east Asian ancestry followed up with de novo genotyping and further replication in east Asian samples provides new insights into blood pressure regulation and potential targets for intervention.
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Genetic associations at 53 loci highlight cell types and biological pathways relevant for kidney function

Cristian Pattaro, +735 more
TL;DR: A meta-analysis of genome-wide association studies for estimated glomerular filtration rate suggests that genetic determinants of eGFR are mediated largely through direct effects within the kidney and highlight important cell types and biological pathways.
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Association of angiotensinogen gene T235 variant with increased risk of coronary heart disease

TL;DR: It is concluded that the T235 polymorphism of the angiotensinogen gene is an independent risk factor, which carries an approximately two-fold increased risk of CHD.