scispace - formally typeset
P

Pavla Stopkova

Researcher at National Institutes of Health

Publications -  67
Citations -  2485

Pavla Stopkova is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Bipolar disorder & Medicine. The author has an hindex of 22, co-authored 59 publications receiving 2140 citations. Previous affiliations of Pavla Stopkova include Charles University in Prague & University of Alabama at Birmingham.

Papers
More filters
Journal ArticleDOI

Assessment of Response to Lithium Maintenance Treatment in Bipolar Disorder: A Consortium on Lithium Genetics (ConLiGen) Report

Mirko Manchia, +104 more
TL;DR: The key phenotypic measures of the “Retrospective Criteria of Long-Term Treatment Response in Research Subjects with Bipolar Disorder” scale currently used in the Consortium on lithium Genetics (ConLiGen) study are reported.
Journal ArticleDOI

Genetic variants associated with response to lithium treatment in bipolar disorder: a genome-wide association study

Liping Hou, +136 more
- 12 Mar 2016 - 
TL;DR: A genome-wide association study of lithium response in 2,563 patients collected by 22 participating sites from the International Consortium on Lithium Genetics (ConLiGen); the largest attempted so far finds a single locus of four linked SNPs on chromosome 21 met genome- wide significance criteria for association with lithium response.
Journal ArticleDOI

Genome-wide association study of 40,000 individuals identifies two novel loci associated with bipolar disorder.

Liping Hou, +148 more
TL;DR: A two-stage meta-analysis of GWAS of bipolar disorder patients and controls revealed genome-wide significant associations at two novel loci, adding to a growing list of common autosomal variants involved in BD and illustrating the power of comparing well-characterized cases to an excess of controls in GWAS.
Journal ArticleDOI

Increase in GSK3β gene copy number variation in bipolar disorder

TL;DR: Patients with BD have an increased frequency of this CNV—primarily the duplication variant—compared with controls, which suggests that GSK3β may be involved in BD susceptibility in some individuals and that CNVs in this and other candidate genes for psychiatric disorders should be analyzed as causative functional genetic variants.
Journal ArticleDOI

Early reduction in prefrontal theta QEEG cordance value predicts response to venlafaxine treatment in patients with resistant depressive disorder.

TL;DR: In this paper, the authors examined whether decrease of prefrontal quantitative EEG (QEEG) cordance value after 1 week of venlafaxine treatment predicts clinical response to Vaxine in resistant patients.