Institution
University of Queensland
Education•Brisbane, Queensland, Australia•
About: University of Queensland is a education organization based out in Brisbane, Queensland, Australia. It is known for research contribution in the topics: Population & Poison control. The organization has 51138 authors who have published 155721 publications receiving 5717659 citations. The organization is also known as: UQ & The University of Queensland.
Papers published on a yearly basis
Papers
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TL;DR: It is shown that the requirements for obtaining secure keys are much easier to meet than for DI-QKD, which opens promising experimental opportunities and clarifies the link between the security of this one-sided DI- QKD scenario and the demonstration of quantum steering, in analogy to the links between DI-ZKD and the violation of Bell inequalities.
Abstract: We analyze the security and feasibility of a protocol for quantum key distribution (QKD) in a context where only one of the two parties trusts his measurement apparatus. This scenario lies naturally between standard QKD, where both parties trust their measurement apparatuses, and device-independent QKD (DI-QKD), where neither do, and can be a natural assumption in some practical situations. We show that the requirements for obtaining secure keys are much easier to meet than for DI-QKD, which opens promising experimental opportunities. We clarify the link between the security of this one-sided DI-QKD scenario and the demonstration of quantum steering, in analogy to the link between DI-QKD and the violation of Bell inequalities.
610 citations
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TL;DR: A previously developed index of walkability was operationalised in an Australian context, using available spatial data to generate a stratified sampling frame for the selection of households for the PLACE study.
609 citations
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TL;DR: While obligate anaerobes have been recovered from carp and tilapia intestines, low ambient temperatures may prevent colonization by anaerOBes in species such as rainbow trout.
Abstract: Bacterial floras isolated from eggs, skin, gills, and intestines have been described for a limited number of fish species. Generally, the range of bacterial genera isolated is related to the aquatic habitat of the fish and varies with factors such as the salinity of the habitat and the bacterial load in the water. In many investigations, identification of isolates to the genus level only makes it difficult to determine the precise relationships of aquatic and fish microfloras. Bacteria recovered from the skin and gills may be transient rather than resident on the fish surfaces. Microfloras of fish intestines appear to vary with the complexity of the fish digestive system. The genera present in the gut generally seem to be those from the environment or diet which can survive and multiply in the intestinal tract, although there is evidence for a distinct intestinal microflora in some species. While obligate anaerobes have been recovered from carp and tilapia intestines, low ambient temperatures may prevent colonization by anaerobes in species such as rainbow trout.
609 citations
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TL;DR: The literature dealing with the electrochemical corrosion characteristics of unalloyed copper in aqueous chloride media is examined in this paper, where a wide range of electrode geometries, the importance of the chloride ion and the mass transport of anodic corrosion products on the corrosion behaviour of copper are made clear for both freshly polished and ‘filmed’ surfaces.
609 citations
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TL;DR: It is shown that a substantial proportion of variation in liability to schizophrenia must be the result of common causal variants, that the variance explained by each chromosome is linearly related to its length, and that the genetic basis of schizophrenia is the same in males and females.
Abstract: Schizophrenia is a complex disorder caused by both genetic and environmental factors. Using 9,087 affected individuals, 12,171 controls and 915,354 imputed SNPs from the Schizophrenia Psychiatric Genome-Wide Association Study (GWAS) Consortium (PGC-SCZ), we estimate that 23% (s.e. = 1%) of variation in liability to schizophrenia is captured by SNPs. We show that a substantial proportion of this variation must be the result of common causal variants, that the variance explained by each chromosome is linearly related to its length (r = 0.89, P = 2.6 × 10 −8 ), that the genetic basis of schizophrenia is the same in males and females, and that a disproportionate proportion of variation is attributable to a set of 2,725 genes expressed in the central nervous system (CNS; P = 7.6 × 10 −8 ). These results are consistent with a
608 citations
Authors
Showing all 52145 results
Name | H-index | Papers | Citations |
---|---|---|---|
Graham A. Colditz | 261 | 1542 | 256034 |
George Davey Smith | 224 | 2540 | 248373 |
David J. Hunter | 213 | 1836 | 207050 |
Daniel Levy | 212 | 933 | 194778 |
Christopher J L Murray | 209 | 754 | 310329 |
Matthew Meyerson | 194 | 553 | 243726 |
Luigi Ferrucci | 193 | 1601 | 181199 |
Nicholas G. Martin | 192 | 1770 | 161952 |
Paul M. Thompson | 183 | 2271 | 146736 |
Jie Zhang | 178 | 4857 | 221720 |
Alan D. Lopez | 172 | 863 | 259291 |
Ian J. Deary | 166 | 1795 | 114161 |
Steven N. Blair | 165 | 879 | 132929 |
Carlos Bustamante | 161 | 770 | 106053 |
David W. Johnson | 160 | 2714 | 140778 |