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Hideaki Konno

Publications -  31
Citations -  8545

Hideaki Konno is an academic researcher. The author has contributed to research in topics: Complementary DNA & cDNA library. The author has an hindex of 19, co-authored 31 publications receiving 8173 citations.

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Journal ArticleDOI

The Transcriptional Landscape of the Mammalian Genome

Piero Carninci, +197 more
- 02 Sep 2005 - 
TL;DR: Detailed polling of transcription start and termination sites and analysis of previously unidentified full-length complementary DNAs derived from the mouse genome provide a comprehensive platform for the comparative analysis of mammalian transcriptional regulation in differentiation and development.
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Analysis of the mouse transcriptome based on functional annotation of 60,770 full-length cDNAs

Yasushi Okazaki, +138 more
- 05 Dec 2002 - 
TL;DR: The present work, completely supported by physical clones, provides the most comprehensive survey of a mammalian transcriptome so far, and is a valuable resource for functional genomics.
Journal ArticleDOI

Genome-wide analysis of mammalian promoter architecture and evolution

TL;DR: These tagging methods allow quantitative analysis of promoter usage in different tissues and show that differentially regulated alternative TSSs are a common feature in protein-coding genes and commonly generate alternative N termini.
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Functional annotation of a full-length mouse cDNA collection

Jun Kawai, +96 more
- 08 Feb 2001 - 
TL;DR: The first RIKEN clone collection is described, which is one of the largest described for any organism and analysis of these cDNAs extends known gene families and identifies new ones.
Journal ArticleDOI

Normalization and subtraction of cap-trapper-selected cDNAs to prepare full-length cDNA libraries for rapid discovery of new genes.

TL;DR: A strategy to prepare normalized and subtracted cDNA libraries in a single step based on hybridization of the first-strand, full-length cDNA with several RNA drivers, including starting mRNA as the normalizing driver and run-off transcripts from minilibraries containing highly expressed genes, rearrayed clones, and previously sequenced cDNAs as subtracting drivers.