M
Mark E. Cooper
Researcher at University of Queensland
Publications - 1514
Citations - 141899
Mark E. Cooper is an academic researcher from University of Queensland. The author has contributed to research in topics: Diabetes mellitus & Diabetic nephropathy. The author has an hindex of 158, co-authored 1463 publications receiving 124887 citations. Previous affiliations of Mark E. Cooper include University of Cambridge & University of Adelaide.
Papers
More filters
Journal ArticleDOI
Membrane binding and perturbation studies of the antimicrobial peptides caerin, citropin, and maculatin.
TL;DR: Data obtained for membrane selectivity using natural lipid extracts show better correlation with minimum inhibitory concentration values against Gram‐positive bacteria and haemolytic activity than that obtained using synthetic DMPG and DMPC.
Journal ArticleDOI
Controls on thallium uptake during hydrothermal alteration of the upper ocean crust
Rosalind M. Coggon,Mark Rehkämper,Charlotte Atteck,Damon A. H. Teagle,Jeffrey C. Alt,Mark E. Cooper +5 more
TL;DR: In this article, the authors investigated the controls on Tl uptake by mid-ocean ridge basalts and evaluate when in the evolution of the ridge flank hydrothermal system Tl-uptake occurs.
Implications of genotype-by-environment interactions for yield adaptation of rainfed lowland rice: influence of flowering date on yield variation
Mark E. Cooper,Boriboon Somrith +1 more
Journal ArticleDOI
Interleukin-1β suppression dampens inflammatory leukocyte production and uptake in atherosclerosis.
Jan Hettwer,Julia Hinterdobler,Benedikt Miritsch,Marcus-André Deutsch,Xinghai Li,Carina Mauersberger,Aldo Moggio,Quinte Braster,Hermann Gram,Avril A. B. Robertson,Mark E. Cooper,Olaf Groß,Markus Krane,Christian Weber,Wolfgang Koenig,Oliver Soehnlein,Oliver Soehnlein,Nicholas H Adamstein,Paul M. Ridker,Heribert Schunkert,Peter Libby,Thorsten Kessler,Hendrik B. Sager +22 more
TL;DR: The authors showed that anti-IL-1β treatment and NLRP3-inflammasome inhibition dampened vascular inflammation and progression of atherosclerosis through reduced blood inflammatory leukocyte 1) supply and 2) uptake into atherosclerotic aortas providing additional mechanistic insights into links between hematopoiesis and atherogenesis.
Journal ArticleDOI
Angiotensin converting enzyme inhibition reduces the expression of transforming growth factor-beta1 and type IV collagen in diabetic vasculopathy.
TL;DR: The concept that TGF-β is involved in the changes associated with diabetic vascular disease is supported, and a mechanism by which angiotensin converting enzyme inhibitors exert their antitrophic effects is suggested.