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Mark E. Cooper

Researcher at University of Queensland

Publications -  1514
Citations -  141899

Mark E. Cooper is an academic researcher from University of Queensland. The author has contributed to research in topics: Diabetes mellitus & Diabetic nephropathy. The author has an hindex of 158, co-authored 1463 publications receiving 124887 citations. Previous affiliations of Mark E. Cooper include University of Cambridge & University of Adelaide.

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Excessive daytime sleepiness increases the risk of motor vehicle crash in obstructive sleep apnea.

TL;DR: The data support the observation that it is those patients with increased sleepiness regardless of OSA severity who are most at risk of motor vehicle crashes, and there is a strong association between excessive daytime sleepiness and increased report of near-misses.
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Silver bullets: A new lustre on an old antimicrobial agent.

TL;DR: This review focuses on the antimicrobial properties of silver and their use in synergistic combination therapy with traditional antibiotic drugs.
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ALT-946 and Aminoguanidine, Inhibitors of Advanced Glycation, Improve Severe Nephropathy in the Diabetic Transgenic (mREN-2)27 Rat

TL;DR: Findings indicate that even in the context of renal injury presumed to be primarily blood pressure- and/or angiotensin II-dependent, approaches that interfere with metabolic pathways such as inhibitors of AGE formation can confer renal protection in experimental diabetes.
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Hypoglycaemia, fear of hypoglycaemia and quality of life in children with Type 1 diabetes and their parents

TL;DR: To evaluate the association between fear of hypoglycaemia, episodes of hyp glucosecaemia and quality of life in children with Type 1 diabetes and their parents, a large number of children and parents with type 1 diabetes were surveyed.
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Dedifferentiation of Immortalized Human Podocytes in Response to Transforming Growth Factor-β: A Model for Diabetic Podocytopathy

TL;DR: Diabetic nephropathy is associated with dedifferentiation of podocytes, losing the specialized features required for efficient glomerular function and acquiring a number of profibrotic, proinflammatory, and proliferative features from tight junction and cytoskeletal rearrangement, augmented proliferation, and apoptosis.