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Per Eriksson

Researcher at Karolinska University Hospital

Publications -  581
Citations -  37340

Per Eriksson is an academic researcher from Karolinska University Hospital. The author has contributed to research in topics: Medicine & Genome-wide association study. The author has an hindex of 92, co-authored 529 publications receiving 34012 citations. Previous affiliations of Per Eriksson include Stavanger University Hospital & Uppsala University.

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Genetic studies of body mass index yield new insights for obesity biology

Adam E. Locke, +481 more
TL;DR: This paper conducted a genome-wide association study and meta-analysis of body mass index (BMI), a measure commonly used to define obesity and assess adiposity, in up to 339,224 individuals.
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Large-scale association analysis identifies new risk loci for coronary artery disease

Panos Deloukas, +204 more
- 01 Jan 2013 - 
TL;DR: An association analysis in CAD cases and controls identifies 15 loci reaching genome-wide significance, taking the number of susceptibility loci for CAD to 46, and a further 104 independent variants strongly associated with CAD at a 5% false discovery rate (FDR).
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Allele-specific increase in basal transcription of the plasminogen-activator inhibitor 1 gene is associated with myocardial infarction

TL;DR: Evidence for an independent, etiological role of PAI-1 in myocardial infarction is provided and the 4G allele of a recently described common 4/5-guanine-tract (4G/5G) polymorphism in the PAi-1 promoter is associated with higher plasma PAI -1 activity.
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A genome-wide association study in Europeans and South Asians identifies five new loci for coronary artery disease

John F. Peden, +110 more
- 01 Apr 2011 - 
TL;DR: Genome-wide association studies have identified 11 common variants convincingly associated with coronary artery disease (CAD), a modest number considering the apparent heritability of CAD(8) as mentioned in this paper.
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Functional Polymorphism in the Regulatory Region of Gelatinase B Gene in Relation to Severity of Coronary Atherosclerosis

TL;DR: Testing the hypothesis that sequence variation in the promoter region of the gelatinase B gene influences its expression, predisposing individuals carrying certain genetic variants to more severe atherosclerosis suggests that this functional genetic variation influences gelatinases B gene promoter activity in an allele-specific manner and has an effect on atherosclerotic phenotype.