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Bruce S. McEwen
Researcher at Rockefeller University
Publications - 1168
Citations - 214913
Bruce S. McEwen is an academic researcher from Rockefeller University. The author has contributed to research in topics: Hippocampus & Hippocampal formation. The author has an hindex of 215, co-authored 1163 publications receiving 200638 citations. Previous affiliations of Bruce S. McEwen include Yale University & National Institutes of Health.
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Journal Article
Stress-induced enhancement of antigen-specific cell-mediated immunity
TL;DR: The results demonstrate that a relatively mild behavioral manipulation can enhance an important class of immune responses that mediate harmful (allergic dermatitis) as well as beneficial (resistance to certain viruses, bacteria, and tumors) aspects of immune function.
Book ChapterDOI
Allostasis and Allostatic Load
TL;DR: Allostasis and allostatic load as discussed by the authors are terms which supplement the classic terms homeostasis, stress, and stress and mediators of allostasis include stress hormones as well as the autonomic nervous systems and pro-inflammatory cytokines and metabolic hormones.
Journal ArticleDOI
Prenatal stress selectively alters the reactivity of the hypothalamic-pituitary adrenal system in the female rat.
TL;DR: The data show that prenatal stress can cause permanent alterations in the behavior of both sexes in stressful situations but appears to cause a selective effect on the HPA axis in the female rat.
Journal ArticleDOI
Stress effects on morphology and function of the hippocampus.
TL;DR: It is discovered that the hippocampus of rodents and tree shrews shows atrophy of pyramidal neurons in the CA3 region, indicating that excitatory amino acid release mediates the atrophy, which likely involves disassembly of the dendritic cytoskeleton.
Journal ArticleDOI
Impact of social environment characteristics on neuroendocrine regulation.
Teresa E. Seeman,Bruce S. McEwen +1 more
TL;DR: Evidence is provided for the hypothesis that observed associations between social ties and health and longevity result, at least partially, from the positive influence of such social environment characteristics in reducing neuroendocrine reactivity.