Bruce S. McEwen

TL;DR: A brief neonatal excess of thyroid hormone produces impairments in spatial learning along with decreases in LTP, long held as a model of learning and memory.

TL;DR: It is suggested that 17beta-estradiol may regulate neuropeptide Y expression mediated by nuclear estrogen receptor alpha-dependent activation in a subset of hippocampal interneurons, and subsequent neuropePTide Y release may indirectly contribute to regulate glutamate-dependent neuronal activity in the adult rat hippocampus.

TL;DR: Among the aged rats in this study with the least retinal degeneration, there was little evidence for a subset of rats that were unable, with extensive training, to learn a platform position, which illustrates the potentially serious confounding effects of deteriorating visual ability on attempts to assess cognitive functioning of aged albino rats on tasks requiring utilization of visual cues.

TL;DR: The neurobiological correlates of individual rats' propensities to develop drug‐intake are enhanced and some putative mechanisms for the dopaminergic hyperactivity that characterizes drug‐prone animals are provided.

TL;DR: These results appear to describe a selective genomic stress response and relate it to human health and disease, particularly stress related maladies such as Post-traumatic Stress Disorder, which have recently been shown to have both epigenetic elements in their causation as well as differences in epigenetic marking of retrotransposons in human patients.