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Bruce S. McEwen

Researcher at Rockefeller University

Publications -  1168
Citations -  214913

Bruce S. McEwen is an academic researcher from Rockefeller University. The author has contributed to research in topics: Hippocampus & Hippocampal formation. The author has an hindex of 215, co-authored 1163 publications receiving 200638 citations. Previous affiliations of Bruce S. McEwen include Yale University & National Institutes of Health.

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Estradiol affects spinophilin protein differently in gonadectomized males and females.

TL;DR: It is reported that E exerts sex-specific effects on dendritic spinophilin-labeled spines in the CA1 region: E treatment significantly increased spinophILin-Ir puncta, indicative of spines, in females, but led to a decrease in males.
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Muscarinic cholinergic receptors in the songbird and quail brain: a quantitative autoradiographic study.

TL;DR: The distribution of muscarinic cholinergic receptors in the brain of three avian species was investigated by quantitative autoradiography and demonstrated that the binding of NMS in the three species is saturable in the nanomolar range and has a high affinity.
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Genomic effects of cold and isolation stress on magnocellular vasopressin mRNA-containing cells in the hypothalamus of the rat.

TL;DR: The results suggest that psychological and metabolic stress may be added to the list of stressors that activate the hypothalamo–neurohypophysial system.
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Metabolic signature in nucleus accumbens for anti-depressant-like effects of acetyl-L-carnitine

TL;DR: A metabolic signature in the NAc for antidepressant-like effects of LAC in vulnerable mice characterized by restoration of stress-induced neuroenergetics alterations and lipid function is revealed.
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Ovarian steroids alter mu opioid receptor trafficking in hippocampal parvalbumin GABAergic interneurons

TL;DR: It is demonstrated that estrogen levels positively regulate the availability of MORs on GABAergic interneurons in the dentate gyrus, suggesting cooperative interaction between opioids and estrogens in modulating principal cell excitability.