Bruce S. McEwen

TL;DR: The excess glucocorticoids and CRH, and the state of anticipatory anxiety, contribute to allostatic load, a new term that refers to the wear and tear on the body and brain arising from attempts to adapt to adversity.

TL;DR: Results indicate that ATD can substantially protect the neonatal rat brain from the differentiative effects of exogenous or endogenous T, probably by blocking aromatization.

TL;DR: In this paper, a combination of electrophysiological, biochemical, and behavioral approaches was used to identify the cellular targets of acute stress in the prefrontal cortex (PFC) and found that behavioral stressors in vivo cause a long-lasting potentiation of NMDAR- and AMPAR-mediated synaptic currents via glucocorticoid receptors selectively in PFC pyramidal neurons.

TL;DR: Stress hormones orchestrate a large-scale redistribution of immune cells in the body and appear to show differential sensitivities and redistribution responses to each hormone depending on the type of leukocyte (neutrophil, monocyte or lymphocyte) and its maturation/functional characteristics.

TL;DR: The current cross-sectional findings support the hypothesis that stress-induced cortisol secretion may contribute to central fat and demonstrate a link between psychological stress and risk for disease.