P
Philip C. Calder
Researcher at University of Southampton
Publications - 808
Citations - 70822
Philip C. Calder is an academic researcher from University of Southampton. The author has contributed to research in topics: Polyunsaturated fatty acid & Eicosapentaenoic acid. The author has an hindex of 125, co-authored 747 publications receiving 59110 citations. Previous affiliations of Philip C. Calder include Southampton General Hospital & Radboud University Nijmegen Medical Centre.
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Journal ArticleDOI
Effects of a fish oil containing lipid emulsion on plasma phospholipid fatty acids, inflammatory markers, and clinical outcomes in septic patients a randomized, controlled clinical trial
TL;DR: Inclusion of fish oil in parenteral nutrition provided to septic ICU patients increases plasma eicosapentaenoic acid, modifies inflammatory cytokine concentrations and improves gas exchange, associated with a tendency towards shorter length of hospital stay.
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Long-chain fatty acids and inflammation
TL;DR: The exposure of inflammatory cells to different types of fatty acids can influence their function and so has the potential to modify inflammatory processes.
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Maternal plasma polyunsaturated fatty acid status in late pregnancy is associated with offspring body composition in childhood.
Rebecca J Moon,Nicholas C. Harvey,Sian M. Robinson,Georgia Ntani,Justin H Davies,Hazel Inskip,Keith M. Godfrey,Keith M. Godfrey,Elaine M. Dennison,Philip C. Calder,Philip C. Calder,Cyrus Cooper,Cyrus Cooper +12 more
TL;DR: This observational study suggests that maternal n-6 PUFA status during pregnancy might influence offspring adiposity in childhood.
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Effects of dairy products naturally enriched with cis-9,trans-11 conjugated linoleic acid on the blood lipid profile in healthy middle-aged men
Sabine Tricon,Graham C. Burdge,Emma L Jones,Jennifer J. Russell,Soraya El-Khazen,Emmanuelle Moretti,Wendy L. Hall,Andrew B. Gerry,David S. Leake,Robert F Grimble,Christine M. Williams,Philip C. Calder,Parveen Yaqoob +12 more
TL;DR: Overall, increased consumption of full-fat dairy products and naturally derived trans fatty acids did not cause significant changes in cardiovascular disease risk variables, as may be expected on the basis of current health recommendations.
Journal Article
The inhibition of T-lymphocyte proliferation by fatty acids is via an eicosanoid-independent mechanism.
TL;DR: Significant evidence is provided that the inhibition of lymphocyte proliferation by fatty acids is independent of the production of eicosanoids, and other mechanisms must be investigated if the effect of fatty acids upon lymphocytes proliferation is to be understood at a biochemical level.