P
Philip C. Calder
Researcher at University of Southampton
Publications - 808
Citations - 70822
Philip C. Calder is an academic researcher from University of Southampton. The author has contributed to research in topics: Polyunsaturated fatty acid & Eicosapentaenoic acid. The author has an hindex of 125, co-authored 747 publications receiving 59110 citations. Previous affiliations of Philip C. Calder include Southampton General Hospital & Radboud University Nijmegen Medical Centre.
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Journal ArticleDOI
High Dose of A Conjugated Linoleic Acid Mixture Increases Insulin Resistance in Rats Fed Either A Low Fat or A High Fat Diet.
Priscila Nogueira Bezan,Héric Holland,Gabriela Salim de Castro,Gabriela Salim de Castro,João Felipe Rito Cardoso,Paula Payão Ovidio,Philip C. Calder,Philip C. Calder,Alceu Afonso Jordão +8 more
TL;DR: It is concluded that a high dose of CLA mixture increases insulin resistance and exacerbates hepatic steatosis when combined with a high fat diet.
Journal ArticleDOI
Effect of changing the lipid component of home parenteral nutrition in adults
S. Osowska,Marek Kunecki,Jacek Sobocki,J. Tokarczyk,K. Majewska,Mohamed Omidi,Marek Radkowski,Helena L. Fisk,Philip C. Calder +8 more
TL;DR: Both SMOFLipid and ClinOleic significantly alter the fatty acid profile of plasma in adult HPN patients previously using Intralipid, and neither LE induces EFA deficiency in these patients.
Journal ArticleDOI
Fish oil supplementation decreases oxidative stress but does not affect platelet-activating factor bioactivity in lungs of asthmatic rats.
A.L. Zanatta,D. T. S. Z. Miranda,B. C. L. Dias,R.M. Campos,M.C. Massaro,Pedro Vicente Michelotto,Annette L. West,Elizabeth A. Miles,Philip C. Calder,Philip C. Calder,Anita Nishiyama +10 more
TL;DR: A dual effect of fish oil on oxidative stress and inflammation in asthma is suggested, since the supplement lowered oxidative stress in the lungs of asthmatic rats, presenting an antioxidant effect, but did not affect PAF bioactivity.
Journal ArticleDOI
Oral administration of EPA-rich oil impairs collagen reorganization due to elevated production of IL-10 during skin wound healing in mice.
Beatriz Burger,Carolina M. C. Kuhl,Thamiris Candreva,Renato da S. Cardoso,Jéssica R. Silva,Bianca Gazieri Castelucci,Sílvio Roberto Consonni,Helena L. Fisk,Philip C. Calder,Philip C. Calder,Marco Aurélio Ramirez Vinolo,Hosana G. Rodrigues +11 more
TL;DR: Oral administration of EPA-rich oil impairs the quality of wound healing without affecting the wound closure time likely due to an elevation of the anti-inflammatory cytokine IL-10.