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Philip C. Calder

Researcher at University of Southampton

Publications -  808
Citations -  70822

Philip C. Calder is an academic researcher from University of Southampton. The author has contributed to research in topics: Polyunsaturated fatty acid & Eicosapentaenoic acid. The author has an hindex of 125, co-authored 747 publications receiving 59110 citations. Previous affiliations of Philip C. Calder include Southampton General Hospital & Radboud University Nijmegen Medical Centre.

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Dietary supplementation with eicosapentaenoic acid, but not with other long-chain n

TL;DR: A moderate amount of EPA but not of other n-6 or n-3 polyunsaturated fatty acids can decrease NK cell activity in healthy subjects.
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Atopy risk in infants and children in relation to early exposure to fish, oily fish, or long-chain omega-3 fatty acids: a systematic review.

TL;DR: It is indicated that provision of fish oil during pregnancy may reduce sensitization to common food allergens and reduce prevalence and severity of atopic dermatitis in the first year of life, with a possible persistence until adolescence with a reduction in eczema, hay fever, and asthma.
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N-3 polyunsaturated fatty acids, inflammation and immunity: pouring oil on troubled waters or another fishy tale?

TL;DR: The immunological effects of large amounts of n -3 PUFA suggest that they might be useful as therapies for diseases characterized by immune dysfunction, and particularly in rheumatoid arthritis and adult asthmatics.
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Marine Omega-3 (N-3) Fatty Acids for Cardiovascular Health: An Update for 2020

TL;DR: This review presents some of the key studies that have investigated EPA and DHA in the primary and secondary prevention of CVD, describes potential mechanisms for their cardioprotective effect, and evaluates the more recently published RCTs in the context of existing scientific literature.
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Lack of effect of foods enriched with plant- or marine-derived n-3 fatty acids on human immune function

TL;DR: An intake of ALA or EPA+DHA does not alter the functional activity of neutrophils, monocytes, or lymphocytes, but it changes the fatty acid composition of mononuclear cells.